ArticlebioRxiv : the preprint server for biology2026
Neuronal expression of Retinoid-Related Orphan Receptor Gamma (RORγ) and revisiting its role in the Central Nervous System.
Article in bioRxiv : the preprint server for biology, 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.
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Abstract
Retinoid-related orphan receptor gamma (RORγ) is a lineage-defining transcription factor for T helper 17 (Th17) cells and has long been considered selectively expressed within the immune system. However, accumulating evidence has implicated Th17 cells and other RORγ-expressing immune populations in embryonic brain development and central nervous system (CNS) function. Notably, RORγ-Cre-mediated deletion of the tuberous sclerosis complex (TSC) has been reported to cause spontaneous seizures and premature lethality, leading to the conclusion that RORγ-expressing immune cells directly regulate CNS physiology. Using multiple RORγ-based reporter mouse lines, we identified unexpected and widespread neuronal labeling throughout the forebrain and cerebellum. RORγ-Cre:GFP
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