ReviewFrontiers in endocrinology2026
Diabetes associated pericyte metabolic signatures and pathogenesis of diabetic retinopathy.
Review in Frontiers in endocrinology, 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 1 paper.
What it found
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The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
The trial behind it
Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.
Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.
Who cites it
1 citing paper in PubMed.
- Endothelial Cell-Pericyte Crosstalk in the Brain Balances Microvascular Stability and Plasticity.Arteriosclerosis, thrombosis, and vascular biology · 2026Review
Corrections and comments
PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.
Authors and funding
3 authors.
Funding
Abstract
Pericytes are metabolically active perivascular supporting cells. They are essential for establishing and maintaining the inner blood retinal barrier and retinal neurovascular homeostasis. In diabetic retinopathy, the chronic hyperglycemic environment adversely affects pericyte functions including their glucose flux and metabolism. The early alterations in pericyte metabolic signatures during diabetes make them susceptible as an early damage target. Altered pericyte glucose flux through the polyol pathway, pentose phosphate pathway, hexosamine biosynthesis pathway, protein kinase C pathway, and advanced glycated end products pathway are responsible for mitochondrial dysfunction, inflammation, and oxidative stress. These metabolic changes in pericytes regulate vascular blood flow and tight junctions' integrity leading to vascular dysfunction and disease progression. This review aims to discuss the current understanding of pericyte metabolic signatures and altered glucose metabolism under hyperglycemic conditions like those present in diabetes. We also aim to highlight the need for further investigation of how pericyte metabolic activities, particularly among pericyte subtypes of the retina and other organs, are coordinated. This knowledge will help to further our understanding of how to preserve the blood tissue barrier and its function, and to prevent not only diabetic retinopathy but also vascular dysfunction in other organs impacted by diabetes.
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Registered trials
Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.