Evidence mapPaperPMID 41869341Full record

ReviewFrontiers in immunology2026

Lactylation modification - a bridge between sepsis and macrophage metabolic reprogramming.

Zhe Fang, Gui Song Zhu, Deng Yun Nie, Biao Xu

Abstract readReview
In one paragraph

Review in Frontiers in immunology, 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 2 papers.

0numbers the graph read from it
0cells of the map it votes in
2citing papers in PubMed
field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

2 citing papers in PubMed.

  1. Review
  2. Review
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

4 authors.

Zhe FangNanjing Hosptial of Chinese Medicine affiliated to Nanjing University of Chinese Medicine, Nanjing, China.
Gui Song ZhuNanjing Hosptial of Chinese Medicine affiliated to Nanjing University of Chinese Medicine, Nanjing, China.
Deng Yun NieNanjing Hosptial of Chinese Medicine affiliated to Nanjing University of Chinese Medicine, Nanjing, China.
Biao XuNanjing Hosptial of Chinese Medicine affiliated to Nanjing University of Chinese Medicine, Nanjing, China.

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Sepsis is a life-threatening organ failure syndrome triggered by dysregulated host responses to infection. During disease progression, macrophages drive initial hyperinflammatory responses and critically regulate the subsequent immunosuppressive phase. Emerging evidence reveals that macrophage phenotypes dynamically adapt through metabolic reprogramming, creating phenotype-metabolism interdependence. Notably, lactate-long considered merely a glycolytic byproduct-now emerges as a key regulator through histone lactylation. This epigenetic modification fine-tunes macrophage functionality and corrects inflammatory imbalance in sepsis. This breakthrough illuminates lactylation's central role in macrophage regulation, opening new diagnostic and therapeutic avenues. This review comprehensively examines lactylation mechanisms and their impact on metabolic control in sepsis-associated macrophages.

Indexed as

Lactic AcidMacrophagesSepsisAnimalsEpigenesis, GeneticGlycolysisHistonesHumansMetabolic ReprogrammingHistonesLactic Acidglycolysislactation modificationmacrophagesmetabolic reprogrammingsepsis

Identifiers

PMID41869341
PMCPMC13002459

What Socratic holds

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LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.