Evidence mapPaperPMID 41876369Full record

ReviewJournal of immunology (Baltimore, Md. : 1950)2026

Vitreoretinal Myeloid Cell Heterogeneity: Diverse Roles in Homeostasis, Immune Surveillance, and Pathophysiology.

Colin A Lemire, Jeremy A Lavine

Abstract readReview
In one paragraph

Review in Journal of immunology (Baltimore, Md. : 1950), 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.

0numbers the graph read from it
0cells of the map it votes in
0citing papers in PubMed
field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

0 citing papers in PubMed.

No citing paper in PubMed yet.

4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

2 authors.

Colin A LemireDepartment of Ophthalmology, Northwestern University Feinberg School of Medicine, Chicago, IL 60611, United States.ORCID 0000-0002-8270-2908
Jeremy A LavineDepartment of Ophthalmology, Northwestern University Feinberg School of Medicine, Chicago, IL 60611, United States.

Funding

Retinal perivascular macrophages: ontology and function during neuroinflammationR01EY036826 · NORTHWESTERN UNIVERSITY · 2025 to 2025
$400k
NEI NIH HHS EY030923NEI NIH HHS EY034486NEI NIH HHS EY036341NEI NIH HHS EY036826NEI NIH HHS R01 EY036826
6 · The paper itself

Abstract

The retina is a central nervous system tissue with immune-privileged status, protected by the blood-retina barrier and maintained by specialized resident tissue macrophages: microglia, hyalocytes, and perivascular macrophages. These cells exhibit specific ontogeny, spatial localization, and immunologic functions. Each population contributes to homeostasis, phagocytosis, and immunoregulation in their local microenvironment. Microglia are the most abundant immune cells in the retina, and their key functions include vascular and synaptic regulation. Hyalocytes are abundant in the vitreous and at the vitreoretinal interface with key functions including vasoregression and maintenance of a clear visual axis. Perivascular macrophages are present along major venules in the superficial retina and may regulate immune cell transendothelial migration. This review summarizes key vitreoretinal myeloid cell functions, compares/contrasts the relative abundances of antigen presentation machinery and T cell costimulatory molecules, and places these expression levels into the context of immune surveillance in the experimental autoimmune uveitis model.

Indexed as

Immunologic SurveillanceMyeloid CellsRetinaVitreous BodyAnimalsAntigen PresentationBlood-Retinal BarrierHomeostasisHumansMacrophagesUveitisantigen presentationinflammationmacrophageMHCIIretina

Identifiers

PMID41876369
PMCPMC13046070

What Socratic holds

Textmetadata
LicenceCC BY-NC
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.