Evidence mapPaperPMID 41879844Full record

ArticleNaunyn-Schmiedeberg's archives of pharmacology2026

Unnatural amino acid compounds as potent multi-target inhibitors of aldose reductase, α-glucosidase, and α-amylase: integrated in vitro, SAR, and molecular dynamics insights.

Serpil Gerni, Cansu Öztürk, Songül Bayrak, Yeliz Demir, Ufuk Atmaca, Dejan Milenković, Dušan Dimić, Ömer İrfan Küfrevioğlu

Abstract read
In one paragraph

Article in Naunyn-Schmiedeberg's archives of pharmacology, 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 1 paper.

0numbers the graph read from it
0cells of the map it votes in
1citing papers in PubMed
field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

1 citing paper in PubMed.

  1. Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

8 authors.

Serpil GerniDepartment of Chemistry, Faculty of Science, Ataturk University, Erzurum, Turkey. gerni-serpil@outlook.com.
Cansu ÖztürkDepartment of Chemistry, Faculty of Science, Ataturk University, Erzurum, Turkey.
Songül BayrakDepartment of Chemistry, Faculty of Science, Ataturk University, Erzurum, Turkey.
Yeliz DemirDepartment of Chemistry, Faculty of Science, Ataturk University, Erzurum, Turkey. yelizdemir2116@gmail.com.
Ufuk AtmacaDepartment of Chemistry, Faculty of Science, Ataturk University, Erzurum, Turkey.
Dejan MilenkovićDepartment of Science, Institute for Information Technologies, University of Kragujevac, Jovana Cvijića Bb, 34000, Kragujevac, Serbia.
Dušan DimićFaculty of Physical Chemistry, University of Belgrade, Studentski Trg 12-16, 11000, Belgrade, Serbia.
Ömer İrfan KüfrevioğluDepartment of Chemistry, Faculty of Science, Ataturk University, Erzurum, Turkey.

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Diabetes mellitus is a multifactorial metabolic disorder in which sustained post-prandial hyperglycaemia and aberrant activation of the polyol pathway contribute to disease progression and long-term complications. Simultaneous modulation of digestive enzymes and aldose reductase (ALR2) therefore represents a rational multitarget therapeutic strategy. In this study, a series of previously reported aryl-substituted unnatural N-methoxysulfonyl β-ketoester derivatives were investigated for their inhibitory potential against ALR2, α-glucosidase, and α-amylase. Compound 1i exhibited the strongest ALR2 inhibition with K

Indexed as

Aldehyde Reductasealpha-AmylasesAmino AcidsEnzyme InhibitorsGlycoside Hydrolase Inhibitorsalpha-GlucosidasesAnimalsHumansMolecular Docking SimulationMolecular Dynamics SimulationStructure-Activity RelationshipAldehyde Reductasealpha-Amylasesalpha-GlucosidasesAmino AcidsEnzyme InhibitorsGlycoside Hydrolase InhibitorsAldose reductaseMolecular dynamicsN-methoxysulfonyl β-ketoesterα-Glucosidase

Identifiers

PMID41879844
PMCPMC13357411

What Socratic holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.