Evidence mapPaperPMID 41901058Full record

ReviewNutrients2026

The Impact of Diet on Long-Term Oncological Outcomes: Investigating Nutritional Mechanisms in Cancer Prevention Management and Prognosis.

Shubana Hayat, Junaid Ahmad, Sara Naeem, Faiza Yaseen, Sania Aamir, Francesca Guida, Livio Luongo, Sabatino Maione

Abstract readReview
In one paragraph

Review in Nutrients, 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.

0numbers the graph read from it
0cells of the map it votes in
0citing papers in PubMed
field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

0 citing papers in PubMed.

No citing paper in PubMed yet.

4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

8 authors.

Shubana HayatDepartment of Experimental Medicine, Università Degli Studi Della Campania "L. Vanvitelli", 80138 Naples, Italy.
Junaid AhmadDepartment of Experimental Medicine and Surgery, University of Rome Tor Vergata, Via Montpellier 1, 00133 Rome, Italy.
Sara NaeemDepartment of Experimental Medicine, Università Degli Studi Della Campania "L. Vanvitelli", 80138 Naples, Italy.
Faiza YaseenDepartment of Experimental Medicine, Università Degli Studi Della Campania "L. Vanvitelli", 80138 Naples, Italy.
Sania AamirDepartment of Experimental Medicine, Università Degli Studi Della Campania "L. Vanvitelli", 80138 Naples, Italy.ORCID 0000-0002-2344-6924
Francesca GuidaDepartment of Experimental Medicine, Università Degli Studi Della Campania "L. Vanvitelli", 80138 Naples, Italy.
Livio LuongoDepartment of Experimental Medicine, Università Degli Studi Della Campania "L. Vanvitelli", 80138 Naples, Italy.
Sabatino MaioneDepartment of Experimental Medicine, Università Degli Studi Della Campania "L. Vanvitelli", 80138 Naples, Italy.

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Long-term oncological outcomes are significantly influenced by dietary patterns and nutritional status. Emerging evidence suggests that specific nutrients and dietary behaviors modulate the biological pathways involved in cancer initiation, progression, and therapeutic response. Understanding these nutritional mechanisms is essential for optimizing cancer prevention strategies, improving treatment efficacy, and enhancing long-term prognosis. Dieting is a modifiable factor influencing cancer prevention, progression, and survivorship. This review is a molecular, clinical, and epidemiological data amalgamation that aims to figure out the first of the three aspects, i.e., how dietary patterns and nutrients affect carcinogenesis, therapeutic tolerance, and long-term outcomes in long-term oncology. The current review moves from diet-dependent core cancer mechanisms that lead cancer pathways through metabolic reprogramming, inflammation, oxidative stress regulation, and epigenetic alterations. Protective dietary patterns, e.g., plant-based, Mediterranean-style, fiber-rich, and omega-3-fed diets, typically provide lower oxidative and inflammatory loads while also facilitating immune surveillance and metabolic stability. Therapy-personalized nutrition that is high in energy-protein and functional foods is instrumental to treatment tolerance, reduction in complication incidence, and cachexia relief. The newest research highlights the significant influence of epigenetic remodeling and the gut-brain-immune axis as the main processes that connect nutrition to tumor behavior and psychosocial outcomes. Translation into clinical practice changes is still dependent on thoughtfully designed trials, the existence of standard guidelines, and the provision of equal access to digital nutrition tools, despite this advancement. Diet is positioned as a low-toxicity co-therapeutic strategy that supports prevention, treatment efficacy, and long-term survivorship.

Indexed as

DietNeoplasmsNutritional StatusEpigenesis, GeneticHumansPrognosisgut microbiota and epigeneticsinflammationlong-term oncologymetabolismnutrition

Identifiers

PMID41901058
PMCPMC13029041

What Socratic holds

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Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.