Evidence map›Paper›PMID 41909199›Full record

ReviewJournal of hepatocellular carcinoma2026

Cancer-Associated Fibroblasts in the Treatment of Intrahepatic Cholangiocarcinoma: Present and Prospects.

Yimeng Yuan, Ruoyu Zhang, Chengkang Qu, Ning Huang, Liming Wang, Mei Liu

Abstract readReview
In one paragraph

Review in Journal of hepatocellular carcinoma, 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.

0numbers the graph read from it
0cells of the map it votes in
0citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

0 citing papers in PubMed.

No citing paper in PubMed yet.

4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

6 authors.

Yimeng Yuan *Department of Hepatobiliary Surgery, National Cancer Center/National Clinical Research Center for Cancer/Cancer Hospital, Chinese Academy of Medical Sciences and Peking Union Medical College, Beijing, People's Republic of China.ORCID 0000-0001-9305-7951
Ruoyu Zhang *Department of Hepatobiliary Surgery, National Cancer Center/National Clinical Research Center for Cancer/Cancer Hospital, Chinese Academy of Medical Sciences and Peking Union Medical College, Beijing, People's Republic of China.ORCID 0000-0002-3396-9307
Chengkang QuDepartment of Hepatobiliary Surgery, National Cancer Center/National Clinical Research Center for Cancer/Cancer Hospital, Chinese Academy of Medical Sciences and Peking Union Medical College, Beijing, People's Republic of China.
Ning HuangDepartment of Hepatobiliary Surgery, National Cancer Center/National Clinical Research Center for Cancer/Cancer Hospital, Chinese Academy of Medical Sciences and Peking Union Medical College, Beijing, People's Republic of China.ORCID 0000-0002-8874-2601
Liming WangDepartment of Hepatobiliary Surgery, National Cancer Center/National Clinical Research Center for Cancer/Cancer Hospital, Chinese Academy of Medical Sciences and Peking Union Medical College, Beijing, People's Republic of China.ORCID 0000-0002-0418-405X
Mei LiuLaboratory of Cell and Molecular Biology & State Key Laboratory of Molecular Oncology, National Cancer Center/National Clinical Research Center for Cancer/Cancer Hospital, Chinese Academy of Medical Sciences and Peking Union Medical College, Beijing, People's Republic of China.ORCID 0000-0002-7364-6318

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Intrahepatic cholangiocarcinoma (ICC) is a rare, highly malignant, and heterogeneous group of primary hepatic adenocarcinomas that differs distinctly from conventional hepatocellular carcinoma. A specific cell population plays a crucial role in ICC named cancer-associated fibroblasts (CAFs). CAFs are involved in the connective tissue proliferative response in ICC, and this response manifests as a dense, fibrocollagen-rich tumor stroma. Thus, although chemotherapy and radiotherapy are the primary approaches to improve survival rates in patients with ICC, the presence of CAFs still render many patients refractory to these therapies. In this article, we explored the complex disease process of ICC in depth, reviewed the origin, heterogeneity, and function of CAFs, and focused on how CAFs subpopulations could be used as biomarkers and contribute to ICC treatment resistance. We also described current breakthroughs in targeting CAFs to overcome cancer treatment resistance and discussed emerging targeted therapies for CAFs. This article aims to provide a comprehensive overview of the latest advances and breakthrough directions in ICC treatment, hoping to offer new insights for future research and clinical practice.

Indexed as

cancer-associated fibroblastscancer therapydrug resistanceintrahepatic cholangiocarcinomaresistance mechanismstumor microenvironment

Identifiers

PMID41909199
PMCPMC13031821

What Socratic holds

Textmetadata
LicenceCC BY-NC
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.