ArticleJournal of veterinary internal medicine2026
Echocardiographic evaluation of pulmonary vascular resistance in 459 dogs with Doppler-derived pulmonary hypertension.
Article in Journal of veterinary internal medicine, 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.
What it found
Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.
The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
The trial behind it
Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.
Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.
Who cites it
0 citing papers in PubMed.
No citing paper in PubMed yet.
Corrections and comments
PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.
Authors and funding
3 authors.
Funding
No grant is acknowledged in the PubMed record.
Abstract
backgroundAbnormally high pulmonary vascular resistance (PVR) is prognostically important and a direct therapeutic target. There is a lack of data on PVR in dogs with pulmonary hypertension (PH). HYPOTHESIS/
objectivesEchocardiographic evaluation of PVR in dogs with PH is feasible and clinically useful for more comprehensive characterization of PH and provides valuable information beyond that derived from Doppler-derived PH assessment alone. ANIMALS: Four hundred fifty-nine client-owned dogs with echocardiographically-determined PH and 38 control dogs.
methodsRetrospective cross-sectional study. Eighteen clinical and 46 echocardiographic variables were evaluated. PVR was calculated using 3 equations validated in people. Dogs with precapillary PH (pre-PH), postcapillary PH (post-PH), isolated post-PH (Ipc-PH), and combined post- and pre-PH (Cpc-PH) were compared using common statistical tests for continuous and categorical data.
resultsThere were 213 dogs with pre-PH and 246 dogs with post-PH (86 with Ipc-PH, 160 with Cpc-PH). Average PVR (mean ± SD or median/range) was 2.5 ± 0.9 WU, 8.8 (1.5-28) WU, 4.6 (1.2-6.1) WU, and 9.2 (6.1-26.8) WU in control dogs and dogs with pre-PH, Ipc-PH, and Cpc-PH, respectively (P < .001). In 28 dogs with post-PH discordant changes of PVR and systolic pulmonary artery pressure (SPAP) were observed. These dogs had either normal or mildly increased SPAP (48 ± 4 mmHg) and high PVR (8.8 ± 2.1 WU) or high SPAP (80 ± 15 mmHg) and normal PVR (4.4 ± 1.2 WU), identifying dogs where estimation of PVR would be most useful. CONCLUSIONS AND CLINICAL IMPORTANCE: Evaluations of PVR might be considered in the clinical assessment of dogs with echocardiographically-determined PH but more study is needed.
Indexed as
Identifiers
What Socratic holds
Registered trials
Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.