Evidence map›Paper›PMID 41914486›Full record

ArticleKorean journal of radiology2026

Prognostic Significance of Pretreatment ¹⁸F-FDG PET/CT Parameters in Patients With ER+/HER2- Metastatic Breast Cancer Treated With CDK4/6 Inhibitors Plus Endocrine Therapy.

Minseung Suh, Jeongryul Ryu, Hojin Song, Jae Ho Jeong, Sangwon Han, Hyehyun Jeong, Jeong Eun Kim, Yeokyeong Shin, Byung-Kwan Jeong, Hee Jin Lee and 5 more

Abstract read
In one paragraph

Article in Korean journal of radiology, 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 1 paper.

0numbers the graph read from it
0cells of the map it votes in
1citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

1 citing paper in PubMed.

  1. Article
4 · The record

Corrections and comments

5 · Who and what money

Authors and funding

15 authors.

Minseung Suh *Department of Oncology, University of Ulsan College of Medicine, Asan Medical Center, Seoul, Republic of Korea.ORCID https://orcid.org/0009-0008-8502-5341
Jeongryul Ryu *Department of Nuclear Medicine, University of Ulsan College of Medicine, Asan Medical Center, Seoul, Republic of Korea.ORCID https://orcid.org/0009-0005-1082-0306
Hojin SongDepartment of Nuclear Medicine, University of Ulsan College of Medicine, Asan Medical Center, Seoul, Republic of Korea.ORCID https://orcid.org/0009-0007-4374-2345
Jae Ho JeongDepartment of Oncology, University of Ulsan College of Medicine, Asan Medical Center, Seoul, Republic of Korea. jaeho.jeong@amc.seoul.kr.ORCID https://orcid.org/0000-0002-8749-2612
Sangwon HanDepartment of Nuclear Medicine, University of Ulsan College of Medicine, Asan Medical Center, Seoul, Republic of Korea. hswon87@naver.com.ORCID https://orcid.org/0000-0001-8095-0396
Hyehyun JeongDepartment of Oncology, University of Ulsan College of Medicine, Asan Medical Center, Seoul, Republic of Korea.ORCID https://orcid.org/0000-0001-7277-6463
Jeong Eun KimDepartment of Oncology, University of Ulsan College of Medicine, Asan Medical Center, Seoul, Republic of Korea.ORCID https://orcid.org/0009-0007-5836-587X
Yeokyeong ShinDepartment of Oncology, University of Ulsan College of Medicine, Asan Medical Center, Seoul, Republic of Korea.ORCID https://orcid.org/0009-0000-0403-2091
Byung-Kwan JeongDepartment of Pathology, University of Ulsan College of Medicine, Asan Medical Center, Seoul, Republic of Korea.ORCID https://orcid.org/0000-0002-0038-3959
Hee Jin LeeDepartment of Pathology, University of Ulsan College of Medicine, Asan Medical Center, Seoul, Republic of Korea.ORCID https://orcid.org/0000-0002-4963-6603
Gyungyub GongDepartment of Pathology, University of Ulsan College of Medicine, Asan Medical Center, Seoul, Republic of Korea.ORCID https://orcid.org/0000-0001-5743-0712
Jin-Hee AhnDepartment of Oncology, University of Ulsan College of Medicine, Asan Medical Center, Seoul, Republic of Korea.ORCID https://orcid.org/0000-0003-4994-9965
Kyung Hae JungDepartment of Oncology, University of Ulsan College of Medicine, Asan Medical Center, Seoul, Republic of Korea.ORCID https://orcid.org/0000-0002-1580-7224
Sung-Bae KimDepartment of Oncology, University of Ulsan College of Medicine, Asan Medical Center, Seoul, Republic of Korea.ORCID https://orcid.org/0000-0001-5588-8332
Dae Hyuk MoonDepartment of Nuclear Medicine, University of Ulsan College of Medicine, Asan Medical Center, Seoul, Republic of Korea.ORCID https://orcid.org/0000-0001-9892-4276

Funding

Korea Health Industry Development Institute RS-2018-KH049510Korea Medical Institute
6 · The paper itself

Abstract

objectiveCyclin-dependent kinase 4/6 (CDK4/6) inhibitors combined with endocrine therapy (ET) constitute the standard systemic treatment for estrogen receptor-positive, human epidermal growth factor 2-negative (ER+/HER2-) metastatic breast cancer (MBC). However, treatment responses remain heterogeneous, highlighting the need for reliable prognostic markers. This study aimed to evaluate the prognostic significance of ¹⁸F-fluorodeoxyglucose (FDG) PET/CT findings in this setting. MATERIALS AND

methodsThis retrospective single-center cohort study included patients with ER+/HER2- MBC who underwent ¹⁸F-FDG PET/CT before initiating CDK4/6 inhibitors plus ET between 2018 and 2023. Maximum standardized uptake value (SUVmax), whole-body metabolic tumor volume (MTV), and total lesion glycolysis (TLG) were measured. Progression-free survival (PFS) and overall survival (OS) were evaluated as the primary and secondary outcomes, respectively, using multivariable Cox models. PET parameters (SUVmax, MTV, and TLG) were analyzed as both continuous and dichotomized variables based on median values, adjusting for relevant clinical covariates.

resultsAmong the 374 patients, 82 (21.9%) presented with de novo metastatic disease, and 357 (95.5%) received CDK4/6 inhibitors as first-line therapy. In multivariable Cox analysis, all continuous PET parameters were independently associated with PFS (adjusted hazard ratio for SUVmax 1.05 [95% confidence interval 1.02-1.08]; log-transformed MTV 1.16 [1.08-1.25]; and log-transformed TLG 1.14 [1.07-1.23]) and OS (SUVmax 1.08 [1.04-1.11]; log-transformed MTV 1.24 [1.12-1.38]; and log-transformed TLG 1.22 [1.11-1.34]) with all

conclusionPretreatment ¹⁸F-FDG PET/CT parameters are independent prognostic markers in patients with ER+/HER2- MBC receiving CDK4/6 inhibitors with ET, supporting their potential utility in risk stratification.

Indexed as

Breast NeoplasmsCyclin-Dependent Kinase 6Positron Emission Tomography Computed TomographyProtein Kinase InhibitorsAdultAgedAntineoplastic Agents, HormonalCyclin-Dependent Kinase 4Erb-b2 Receptor Tyrosine KinasesFemaleFluorodeoxyglucose F18HumansMiddle AgedPrognosisRadiopharmaceuticalsReceptors, EstrogenAntineoplastic Agents, HormonalCDK6 protein, humanCyclin-Dependent Kinase 4Cyclin-Dependent Kinase 6Erb-b2 Receptor Tyrosine KinasesFluorodeoxyglucose F18Protein Kinase InhibitorsRadiopharmaceuticalsReceptors, Estrogen¹⁸F-FDG PETBreast cancerCDK4/6 inhibitorMetastasisPrognostic biomarker

Identifiers

PMID41914486
PMCPMC13056453

What Socratic holds

Textmetadata
LicenceCC BY-NC
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.