ArticleEuropean journal of clinical nutrition2026
Sugar-sweetened beverages and acute metabolic crises in type 2 diabetes mellitus: a report of two cases.
Article in European journal of clinical nutrition, 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.
What it found
Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.
The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
The trial behind it
Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.
Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.
Who cites it
0 citing papers in PubMed.
No citing paper in PubMed yet.
Corrections and comments
PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.
Authors and funding
6 authors.
Funding
No grant is acknowledged in the PubMed record.
Abstract
Diabetic ketoacidosis (DKA) is an uncommon but serious complication in patients with type 2 diabetes mellitus (T2DM). Excessive intake of sugar-sweetened beverages (SSBs) may represent an underrecognized trigger for DKA in individuals with T2DM. We present two male patients with T2DM who developed ketosis following excessive consumption of SSBs (aerated drinks). The first patient, aged 33 years, reported consuming 2.5 litre/day of SSBs and developed ketosis without acidosis. The second, aged 18 years, reported daily intake of 4 litre/day of SSBs and presented with frank DKA. Both patients were managed with insulin therapy, which was successfully tapered off post-discharge. At follow-up, glycemic control was maintained with oral antidiabetic therapy or lifestyle modification alone. Both patients exhibited clinical signs of insulin resistance and were negative for anti-glutamic acid decarboxylase antibodies. These cases underscore the potential role of SSBs in precipitating ketosis in T2DM patients.
Indexed as
Identifiers
41917428What Socratic holds
Registered trials
Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.