ArticleJournal of anesthesia and translational medicine2024
Mapping the publication trends of synaptic plasticity in neuropathic pain: A bibliometric analysis.
Article in Journal of anesthesia and translational medicine, 2024. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.
What it found
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The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
The trial behind it
Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.
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Who cites it
0 citing papers in PubMed.
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Corrections and comments
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Authors and funding
7 authors.
Funding
No grant is acknowledged in the PubMed record.
Abstract
Background: Neuropathic pain typically arises following injury or damage to the somatic nervous system, a chronic and debilitating condition with complex mechanisms. Synaptic plasticity, the changes in neuronal connection strength, has long been recognized to participate in the progression of neuropathic pain. Nevertheless, there has been a lack of bibliometric research on synaptic plasticity in neuropathic pain. Methods: A total of 688 papers were retrieved from the WoSCC databases and the software of Citespace, VOSviewer1.6.18 and the Online Analysis Platform of Literature Metrology (https://bibliometric.com/) were used to conduct bibliometric analysis. Results: The study provided an overview of research in the field and identified the most productive countries, institutions and authors. By analyzing the timelines of co-cited reference clusters and the citation bursts of co-cited references and keywords, it is evident that glia-neuron interaction and extracellular matrix mediated synaptic plasticity in neuropathic pain. Additionally, the up-to-date research trend and the constantly being discovered neurotransmitter and receptors were worth more attention. Two recent focal points in this field include microglia-related neuroinflammation and the central nucleus of the amygdala (CeA) plasticity in pain related emotional distress. Conclusions: The study provides a comprehensive overview of the research on neuropathic pain and synaptic plasticity through bibliometrics and knowledge-map analyses. It aims to offer an updated perspective and suggest future directions for scientific researchers in further investigations.
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Registered trials
Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.