Evidence map›Paper›PMID 41929520›Full record

ReviewFrontiers in immunology2026

The role of tryptophan-AhR signaling in the pathogenesis of metabolic dysfunction-associated steatotic liver disease (MASLD): implications for therapeutic strategies.

Yinan Zhao, Shuhao Cheng, Jiu Liang, Guoying Yu

Abstract readReview
In one paragraph

Review in Frontiers in immunology, 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 1 paper.

0numbers the graph read from it
0cells of the map it votes in
1citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

1 citing paper in PubMed.

  1. Review
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

4 authors.

Yinan ZhaoClinical Medical College of Qinghai University, Xining, China.
Shuhao ChengClinical Medical College of Qinghai University, Xining, China.
Jiu LiangClinical Medical College of Qinghai University, Xining, China.
Guoying YuDepartment of Hepatology II, Fourth People's Hospital of Qinghai Province, Xining, China.

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Metabolic dysfunction-associated steatotic liver disease (MASLD) has emerged as a significant global health concern, closely associated with metabolic syndrome and characterized by hepatic fat accumulation, inflammation, and fibrosis. While the pathogenesis of MASLD is multifactorial, recent research has highlighted the role of the tryptophan-aryl hydrocarbon receptor (AhR) signaling pathway in influencing both immune and metabolic functions in the liver. Tryptophan, an essential amino acid, is metabolized into various bioactive metabolites, such as kynurenine, that activate AhR. This activation modulates cellular processes including inflammation, oxidative stress, and lipid metabolism. Emerging evidence suggests that dysregulated tryptophan metabolism and AhR signaling contribute to the progression of MASLD, particularly through immune modulation and alterations in metabolic pathways. This perspective aims to provide an overview of the current understanding of tryptophan-AhR signaling in MASLD, discussing its potential as a therapeutic target and the challenges associated with targeting this pathway. Future research directions are proposed to explore how modulation of the tryptophan-AhR axis could offer novel therapeutic strategies for MASLD, providing new insights into its treatment and management.

Indexed as

Basic Helix-Loop-Helix ProteinsFatty LiverNon-alcoholic Fatty Liver DiseaseReceptors, Aryl HydrocarbonSignal TransductionTryptophanAnimalsHumansKynurenineLiverOxidative StressBasic Helix-Loop-Helix ProteinsKynurenineReceptors, Aryl HydrocarbonTryptophanAhRimmune modulationinflammationkynureninelipid metabolismMASLDtherapeutic strategiestryptophan metabolism

Identifiers

PMID41929520
PMCPMC13038574

What Socratic holds

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Registered trials

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Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.