ReviewFrontiers in immunology2026
The role of tryptophan-AhR signaling in the pathogenesis of metabolic dysfunction-associated steatotic liver disease (MASLD): implications for therapeutic strategies.
Review in Frontiers in immunology, 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 1 paper.
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The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
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Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.
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Who cites it
1 citing paper in PubMed.
- Gut microbiota-immune crosstalk in osteoarthritis: pathogenic mechanisms and emerging therapeutic opportunities.Frontiers in microbiology · 2026Review
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4 authors.
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Abstract
Metabolic dysfunction-associated steatotic liver disease (MASLD) has emerged as a significant global health concern, closely associated with metabolic syndrome and characterized by hepatic fat accumulation, inflammation, and fibrosis. While the pathogenesis of MASLD is multifactorial, recent research has highlighted the role of the tryptophan-aryl hydrocarbon receptor (AhR) signaling pathway in influencing both immune and metabolic functions in the liver. Tryptophan, an essential amino acid, is metabolized into various bioactive metabolites, such as kynurenine, that activate AhR. This activation modulates cellular processes including inflammation, oxidative stress, and lipid metabolism. Emerging evidence suggests that dysregulated tryptophan metabolism and AhR signaling contribute to the progression of MASLD, particularly through immune modulation and alterations in metabolic pathways. This perspective aims to provide an overview of the current understanding of tryptophan-AhR signaling in MASLD, discussing its potential as a therapeutic target and the challenges associated with targeting this pathway. Future research directions are proposed to explore how modulation of the tryptophan-AhR axis could offer novel therapeutic strategies for MASLD, providing new insights into its treatment and management.
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