Evidence mapPaperPMID 41947028Full record

ArticleCPT: pharmacometrics & systems pharmacology2026

The Role of Estradiol in Oxidative Stress in Health and Cystathionine ß-Synthase Deficiency: A Mechanistic Model.

Allison Cruikshank, H Frederik Nijhout, Michael C Reed

Abstract read
In one paragraph

Article in CPT: pharmacometrics & systems pharmacology, 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.

0numbers the graph read from it
0cells of the map it votes in
0citing papers in PubMed
field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

0 citing papers in PubMed.

No citing paper in PubMed yet.

4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

3 authors.

Allison CruikshankDepartment of Mathematics, Duke University, Durham, North Carolina, USA.ORCID https://orcid.org/0000-0002-4780-4984
H Frederik NijhoutDepartment of Biology, Duke University, Durham, North Carolina, USA.
Michael C ReedDepartment of Mathematics, Duke University, Durham, North Carolina, USA.ORCID https://orcid.org/0000-0002-1051-1811

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Oxidative stress occurs when there is an imbalance between oxidants and antioxidants, leading to the accumulation of reactive oxygen species (ROS). Excessive ROS can damage lipids, proteins, and DNA, contributing to cellular dysfunction and disease. Interestingly, premenopausal females tend to have lower levels of oxidative stress and higher concentrations of certain antioxidants, such as glutathione (GSH), compared to males and postmenopausal females. These differences point to the important role of sex hormones in regulating oxidative stress and its effects on the body. Yet, clinical and experimental studies report conflicting effects of estradiol, particularly across the menstrual cycle in premenopausal females and in estradiol supplementation in postmenopausal females. Here, we present a mechanistic mathematical model of hydrogen peroxide (H

Indexed as

EstradiolHomocystinuriaOxidative StressCystathionine beta-SynthaseFemaleHumansHydrogen PeroxideMaleMenstrual CycleModels, BiologicalProgesteroneReactive Oxygen SpeciesCystathionine beta-SynthaseEstradiolHydrogen PeroxideProgesteroneReactive Oxygen SpeciesCBS deficiencyestrogen supplementationoxidative stresssex differences

Identifiers

PMID41947028
PMCPMC13056701

What Socratic holds

Textmetadata
LicenceCC BY-NC
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Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.