Evidence mapPaperPMID 41969200Full record

ArticleDiabetes, obesity & metabolism2026

Association of Semaglutide Treatment With Liver Cirrhosis and Hepatocellular Carcinoma in Type 2 Diabetes: A Population-Based Cohort Study.

Assaf Issachar, Talish Razi, Ilya Borochov, Hadar Duskin Bitan, Ronen Arbel

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Article in Diabetes, obesity & metabolism, 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.

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1 · What the graph read from it

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4 · The record

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5 · Who and what money

Authors and funding

5 authors.

Assaf IssacharLiver Institute, Rabin Medical Center, Beilinson Hospital, Petah Tikva, Israel.ORCID https://orcid.org/0000-0002-7004-7897
Talish RaziCommunity Medical Services Division, Clalit Health Services, Tel Aviv, Israel.ORCID https://orcid.org/0009-0006-2172-1044
Ilya BorochovCommunity Medical Services Division, Clalit Health Services, Tel Aviv, Israel.
Hadar Duskin BitanGray School of Medicine, Tel Aviv University, Tel Aviv, Israel.ORCID https://orcid.org/0000-0002-1721-1360
Ronen ArbelCommunity Medical Services Division, Clalit Health Services, Tel Aviv, Israel.ORCID https://orcid.org/0000-0002-6058-8665

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

backgroundMetabolic dysfunction-associated steatotic liver disease is common among individuals with type 2 diabetes mellitus and substantially increases the risk of liver cirrhosis and hepatocellular carcinoma. Effective therapies that improve metabolic control while preventing advanced liver outcomes are limited.

aimsTo evaluate the association between semaglutide treatment and the risk of liver cirrhosis and hepatocellular carcinoma in adults with type 2 diabetes mellitus.

methodsWe performed a retrospective population-based cohort study using electronic health records from a large integrated healthcare organization. Adults with type 2 diabetes mellitus and no prior diagnosis of liver cirrhosis or hepatocellular carcinoma were included. Exposure was defined as treatment with semaglutide for at least four consecutive months compared with matched control patients. Cox proportional hazards models estimated adjusted hazard ratios with 95% confidence intervals, accounting for demographic factors, body mass index, smoking status, glycaemic control, other antidiabetic treatments and baseline liver fibrosis risk.

resultsAmong 71 612 individuals, 35 806 received semaglutide and 35 806 were matched controls. Over a median follow-up of 4628 days for liver cirrhosis and 4090 days for HCC, 245 patients developed liver cirrhosis and 21 developed hepatocellular carcinomas. In the Cox model, there was no association between semaglutide treatment and the diagnosis of liver cirrhosis (HR 1.3, 95% CI 0.98-1.77) or hepatocellular carcinoma (HR 0.6, 95% CI 0.21-1.51).

conclusionsIn this real-world study, semaglutide treatment was not associated with a reduced risk of liver cirrhosis or hepatocellular carcinoma among adults with type 2 diabetes mellitus.

Indexed as

Carcinoma, HepatocellularDiabetes Mellitus, Type 2Hypoglycemic AgentsLiver CirrhosisLiver NeoplasmsSemaglutideAgedCohort StudiesFemaleHumansMaleMiddle AgedRetrospective StudiesHypoglycemic AgentsSemaglutideglucagon‐like peptide‐1 receptor agonistshepatocellular carcinomaliver cirrhosismetabolic liver diseasereal‐world datasemaglutidetype 2 diabetes mellitus

Identifiers

PMID41969200
PMCPMC13243961

What Socratic holds

Texttitle and abstract
LicenceCC BY-NC
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Registered trials

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Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.