Evidence mapPaperPMID 41973649Full record

ReviewAdipocyte2026

The role and mechanism of UPRmt in adipocytes.

Hao Liu, Jie Chen, Dan-Qi Qiu, Miao-Wei Jiang, Hao-Qi Chen, Li Li, Shu-Qin Chen

Abstract readReview
In one paragraph

Review in Adipocyte, 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.

0numbers the graph read from it
0cells of the map it votes in
0citing papers in PubMed
field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

0 citing papers in PubMed.

No citing paper in PubMed yet.

4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

7 authors.

Hao LiuDepartment of Endocrinology, Yuehu Campus, The First Affiliated Hospital of Ningbo University, Ningbo, People's Republic of China.
Jie ChenDepartment of Endocrinology, Yuehu Campus, The First Affiliated Hospital of Ningbo University, Ningbo, People's Republic of China.
Dan-Qi QiuDepartment of Endocrinology, Yuehu Campus, The First Affiliated Hospital of Ningbo University, Ningbo, People's Republic of China.
Miao-Wei JiangDepartment of Endocrinology, Yuehu Campus, The First Affiliated Hospital of Ningbo University, Ningbo, People's Republic of China.
Hao-Qi ChenDepartment of Endocrinology, Yuehu Campus, The First Affiliated Hospital of Ningbo University, Ningbo, People's Republic of China.
Li LiDepartment of Endocrinology, Yuehu Campus, The First Affiliated Hospital of Ningbo University, Ningbo, People's Republic of China.
Shu-Qin ChenDepartment of Endocrinology, Yuehu Campus, The First Affiliated Hospital of Ningbo University, Ningbo, People's Republic of China.

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Obesity is one of the most significant health challenges today, with its prevalence increasing rapidly worldwide. The associated inflammatory state is a major risk factor for developing type 2 diabetes, cardiovascular diseases, and sleep apnoea, putting immense pressure on global healthcare systems. Abnormal accumulation or dysfunction of adipose tissue can lead to obesity, which is a major risk factor for metabolic and cardiovascular diseases. The mitochondrial unfolded protein response (UPRmt) serves as a critical adaptive mechanism that safeguards cellular homoeostasis during mitochondrial proteostatic stress by orchestrating the expression of chaperones, proteases, and metabolic regulators to restore protein folding capacity and mitigate organelle dysfunction. This review discusses the role of UPRmt in adipocytes, a key player in maintaining metabolic homoeostasis and thermogenesis. Understanding UPRmt's mechanisms could offer novel therapeutic strategies to combat obesity and its complications.

Indexed as

AdipocytesMitochondriaUnfolded Protein ResponseAnimalsHumansObesityadipocytemetabolismMitochondrial unfolded protein responseUPRmt

Identifiers

PMID41973649
PMCPMC13078241

What Socratic holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.