Evidence mapPaperPMID 41998699Full record

ReviewCell communication and signaling : CCS2026

Triggering receptor expressed on myeloid cell family in atherosclerosis: from mechanisms to intervention strategies.

Shu-Jia Gui, Xiao-Wen Gao, Ping Zhang, Yong-Hong Tang, Jia-Hui Gao

Abstract readReview
In one paragraph

Review in Cell communication and signaling : CCS, 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.

0numbers the graph read from it
0cells of the map it votes in
0citing papers in PubMed
field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

0 citing papers in PubMed.

No citing paper in PubMed yet.

4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

5 authors.

Shu-Jia Gui *The Affiliated Nanhua Hospital, Department of Neurology, Hengyang Medical School, University of South China, Hengyang, Hunan, 421001, China.
Xiao-Wen Gao *The Affiliated Nanhua Hospital, Department of Spine surgery, Hengyang Medical School, University of South China, Hengyang, Hunan, 421001, China.
Ping ZhangThe Affiliated Nanhua Hospital, Sleep Medical Center, Hengyang Medical School, University of South China, Hengyang, Hunan, 421001, China.
Yong-Hong TangThe Affiliated Nanhua Hospital, Sleep Medical Center, Hengyang Medical School, University of South China, Hengyang, Hunan, 421001, China. tyh6246@163.com.
Jia-Hui GaoThe Affiliated Nanhua Hospital, Sleep Medical Center, Hengyang Medical School, University of South China, Hengyang, Hunan, 421001, China. lilygao1995@163.com.

Funding

Natural Science Foundation of Hunan Province 2025JJ60778
6 · The paper itself

Abstract

Atherosclerosis is the leading cause of coronary artery disease (CAD). Triggering receptor expressed on myeloid cell (TREM) family members are critical regulators of lipid metabolism, inflammation and extracellular matrix metabolism during atherogenesis and thereby influence foam cell formation, vascular inflammation and plaque stability. Furthermore, soluble TREM proteins arising from the alternative splicing of TREM family mRNAs, proteolytic shedding or single nucleotide polymorphisms, hold significant diagnostic and prognostic value for CAD patients. Therefore, the importance of the TREM family in the prevention and treatment of atherosclerosis should not be underestimated. This review emphasizes the contribution of the TREM family to the pathogenesis of atherosclerosis to provide a comprehensive understanding of the roles of the TREM family in atherosclerosis. Moreover, this review summarizes the clinical significance of soluble TREM proteins in cardiovascular diseases, thereby highlighting their emerging potential as biomarkers in CAD. Finally, the molecular mechanisms regulating TREM family expression and the translational potential of strategies targeting TREM family members in atherosclerosis-associated diseases are discussed.

Indexed as

AtherosclerosisMyeloid CellsReceptors, ImmunologicAnimalsHumansReceptors, ImmunologicAtherosclerosisInflammationLipid metabolismPlaque stabilityTREM family

Identifiers

PMID41998699
PMCPMC13255405

What Socratic holds

Textmetadata
LicenceCC BY-NC-ND
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.