Evidence map›Paper›PMID 42019947›Full record

ReviewImmunoHorizons2026

Cellular and molecular interactions underlying the peripheral immune responses to blood-stage malaria infections.

Kieran Tebben, Rosita Asawa, David Serre, Kirsten E Lyke

Abstract readReview
PubMed Full text at PMC Publisher
In one paragraph

Review in ImmunoHorizons, 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.

0numbers the graph read from it
0cells of the map it votes in
0citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

0 citing papers in PubMed.

No citing paper in PubMed yet.

4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

4 authors.

Kieran TebbenInstitute for Genome Sciences, University of Maryland School of Medicine, Baltimore, MD, United States.ORCID 0000-0001-8866-7667
Rosita AsawaInstitute for Genome Sciences, University of Maryland School of Medicine, Baltimore, MD, United States.
David SerreInstitute for Genome Sciences, University of Maryland School of Medicine, Baltimore, MD, United States.
Kirsten E LykeMalaria Research Program, Center for Vaccine Development and Global Health, University of Maryland School of Medicine, Baltimore, MD, United States.

Funding

Host and Parasite Transcriptional Changes Associated with Immunity to Clinical Malaria in Malian ChildrenR21AI146853 · NIAID · UNIVERSITY OF MARYLAND BALTIMORE · PI SERRE, DAVID, TRAVASSOS, MARK A · 2019 to 2020
$425k
NIH HHS R21AI146853
6 · The paper itself

Abstract

Malaria, caused by Plasmodium species, is one of the most widespread illnesses globally, affecting millions of individuals each year. The complex life cycle of these parasites requires a multifaceted approach from the human immune system to respond to infection. Additionally, Plasmodium parasites have coevolved in primates and developed numerous immune evasion mechanisms to escape human immune defenses. Here, we provide an up-to-date review of the human immune responses to blood-stage malaria, as well as the parasite immune evasion mechanisms during this part of the life cycle.

Indexed as

Host-Parasite InteractionsImmune EvasionMalariaPlasmodiumAnimalsHumansLife Cycle Stageshost-pathogen interactionsimmunologymalaria

Identifiers

PMID42019947
PMCPMC13102483

What Socratic holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.