Evidence mapPaperPMID 42074230Full record

ReviewInternational journal of molecular sciences2026

Hemorheological Alterations as a Driver of Microangiopathy in Diabetic Kidney Disease-The Role of Erythrocyte.

Michael Garoufis, Christina Kostara, Sissy Foteini Sakkou, Sempastian Filippas-Ntekouan, Eleni Bairaktari, Vasileios Tsimihodimos

Abstract readReview
In one paragraph

Review in International journal of molecular sciences, 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.

0numbers the graph read from it
0cells of the map it votes in
0citing papers in PubMed
field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

0 citing papers in PubMed.

No citing paper in PubMed yet.

4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

6 authors.

Michael GaroufisLaboratory of Clinical Chemistry, Faculty of Medicine, School of Health Sciences, University of Ioannina, 45500 Ioannina, Greece.ORCID 0009-0002-3214-8504
Christina KostaraLaboratory of Clinical Chemistry, Faculty of Medicine, School of Health Sciences, University of Ioannina, 45500 Ioannina, Greece.ORCID 0000-0001-7045-1323
Sissy Foteini SakkouDepartment of Internal Medicine, Faculty of Medicine, School of Health Sciences, University of Ioannina, 45500 Ioannina, Greece.
Sempastian Filippas-NtekouanDepartment of Internal Medicine, Faculty of Medicine, School of Health Sciences, University of Ioannina, 45500 Ioannina, Greece.ORCID 0000-0001-7173-5352
Eleni BairaktariLaboratory of Clinical Chemistry, Faculty of Medicine, School of Health Sciences, University of Ioannina, 45500 Ioannina, Greece.
Vasileios TsimihodimosDepartment of Internal Medicine, Faculty of Medicine, School of Health Sciences, University of Ioannina, 45500 Ioannina, Greece.ORCID 0000-0003-1708-3415

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Diabetic kidney disease (DKD) is a severe microvascular complication traditionally attributed to general metabolic derangement and genetic susceptibility. However, this classic pathophysiological approach overlooks the role of red blood cells in the development and the progression of the disease. Prolonged exposure to high blood glucose and oxidative stress compromises the cell's membrane architecture and ionic homeostasis, resulting in altered rheological properties. By synthesizing these molecular-to-rheological pathways, this review establishes a novel pathophysiological framework for understanding DKD, repositioning erythrocytes to a primary catalyst of renal injury and a highly sensitive target for early diagnostic intervention.

Indexed as

Diabetic NephropathiesErythrocytesHemorheologyAnimalsHumansOxidative Stressdiabetic kidney diseaseerythrocytehemorheologymicrovascular dysfunction

Identifiers

PMID42074230
PMCPMC13116247

What Socratic holds

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Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.