Evidence map›Paper›PMID 42074485›Full record

ReviewGenes2026

CNDP1 and Diabetic Kidney Disease: From Genetic Susceptibility to Therapeutic Targeting.

Bulent Tolga Delibasi, Michael Ismail Sarisen, Matthew Thomas Belitsos, Halil Kutlu Erol, Tuncay Delibasi

Abstract readReview
In one paragraph

Review in Genes, 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.

0numbers the graph read from it
0cells of the map it votes in
0citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

0 citing papers in PubMed.

No citing paper in PubMed yet.

4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

5 authors.

Bulent Tolga DelibasiLong School of Medicine, University of Texas Health Science Center San Antonio, San Antonio, TX 78229, USA.ORCID 0009-0004-6410-8536
Michael Ismail SarisenDepartment of Biology, Health, and the Environment, University of Texas, San Antonio, TX 78249, USA.
Matthew Thomas BelitsosDepartment of Biology, Health, and the Environment, University of Texas, San Antonio, TX 78249, USA.
Halil Kutlu ErolDepartment of Medicine/Nephrology, University of Arizona College of Medicine, Tucson, AZ 85724, USA.ORCID 0000-0001-5760-4276
Tuncay DelibasiDepartment of Medicine/Endocrinology, SUNY Upstate Medical University, Syracuse, NY 13210, USA.ORCID 0000-0002-5780-8209

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Diabetic kidney disease (DKD) affects a substantial proportion of individuals with diabetes mellitus and represents the leading cause of end-stage renal disease worldwide. Familial aggregation studies consistently demonstrate that genetic factors contribute significantly to DKD susceptibility beyond metabolic and hemodynamic determinants. The carnosine dipeptidase 1 (

Indexed as

Diabetic NephropathiesDipeptidasesGenetic Predisposition to DiseaseAnimalsCarnosineHumansCarnosineCNDP1 protein, humanDipeptidasescarnosinasecarnosinecarnosine metabolismCNDP1CTG repeat polymorphismdiabetic kidney diseasediabetic nephropathygenetic susceptibilityMannheim variantprecision medicine

Identifiers

PMID42074485
PMCPMC13116943

What Socratic holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.