Evidence map›Paper›PMID 42079641›Full record

ArticleFrontiers in immunology2026

NOD1 modulates chronic obstructive pulmonary disease progression via FOXA1/NLRP3-mediated regulation of pyroptosis.

Wenzhi Xie, Hui Xu, Hong-Mei Shu

Abstract read
In one paragraph

Article in Frontiers in immunology, 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.

0numbers the graph read from it
0cells of the map it votes in
0citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

0 citing papers in PubMed.

No citing paper in PubMed yet.

4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

3 authors.

Wenzhi Xie *Geriatric Department of Anqing Municipal Hospital, Anqing, Anhui, China.
Hui Xu *Department of Internal Medicine (Respiratory Medicine), Wannan Medical University, Wuhu, Anhui, China.
Hong-Mei ShuDepartment of Pulmonary and Critical Care Medicine, Anqing Municipal Hospital, Anqing, Anhui, China.

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Chronic obstructive pulmonary disease (COPD) is characterized by persistent airway inflammation and progressive airflow limitation. In this study, we established cellular models of COPD using bronchial epithelial cells and also constructed animal models to verify the protective effect of NOD1 knockout on lung tissue in COPD model mice, aiming to elucidate the molecular events linking increased NOD1 expression to inflammatory cell death. Transcriptomic and functional analyses revealed that NOD1 promotes pyroptosis in COPD via the FOXA1-NLRP3 signaling axis, with the PI3K-Akt pathway mediating these effects. Mechanistically, NOD1 suppresses FOXA1, leading to upregulation of NLRP3 and enhanced release of pro-inflammatory cytokines IL-18 and IL-1β. Knockdown of NOD1 alleviated pyroptosis and improved cell survival, effects reversed by NLRP3 overexpression. In conclusion, our findings identify the NOD1-FOXA1-NLRP3 axis as a key driver of inflammatory cell death in COPD, advancing our understanding of disease pathogenesis and highlighting potential molecular targets for therapeutic intervention.

Indexed as

Hepatocyte Nuclear Factor 3-alphaNLR Family, Pyrin Domain-Containing 3 ProteinNod1 Signaling Adaptor ProteinPulmonary Disease, Chronic ObstructivePyroptosisAnimalsDisease Models, AnimalDisease ProgressionHumansMaleMiceMice, KnockoutSignal TransductionFOXA1 protein, humanHepatocyte Nuclear Factor 3-alphaNLR Family, Pyrin Domain-Containing 3 ProteinNLRP3 protein, humanNOD1 protein, humanNod1 Signaling Adaptor Proteinchronic obstructive pulmonary disease (COPD)FOXA1-NLRP3 signaling axisinflammatory cell deathNOD1pyroptosis

Identifiers

PMID42079641
PMCPMC13134683

What Socratic holds

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Registered trials

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Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.