Evidence mapPaperPMID 42100413Full record

ReviewFrontiers in oncology2026

Metabolic symbiosis and competition: the dual nature of TAM-tumor cell cross-talk in tumor progression.

Chaokai Ba, Shizheng Tong, Zefan Wang, Huiting Zhu, Shenxian Qian

Abstract readReview
In one paragraph

Review in Frontiers in oncology, 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.

0numbers the graph read from it
0cells of the map it votes in
0citing papers in PubMed
field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

0 citing papers in PubMed.

No citing paper in PubMed yet.

4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

5 authors.

Chaokai Ba *The Fourth School of Clinical Medicine, Zhejiang Chinese Medical University, Hangzhou, China.
Shizheng Tong *The Fourth School of Clinical Medicine, Zhejiang Chinese Medical University, Hangzhou, China.
Zefan Wang *Hangzhou First People's Hospital, Westlake University, Hangzhou, China.
Huiting Zhu *Hangzhou First People's Hospital, Westlake University, Hangzhou, China.
Shenxian QianThe Fourth School of Clinical Medicine, Zhejiang Chinese Medical University, Hangzhou, China.

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Cancer cells and tumor associated macrophages (TAMs) engage in a sophisticated metabolic symbiosis within the tumor microenvironment (TME), where reciprocal metabolite exchange drives immune evasion and malignant progression. This review posits that TAMs functional plasticity is not merely a consequence but a driver of tumor fitness, governed by extensive metabolic rewiring. We dissect the mechanistic underpinnings of this "metabolic dialogue," focusing on the convergence of glycolytic flux, the lactate shuttle, amino acid catabolism, lipid reprogramming, hypoxia-induced adaptations, and TCA cycle anaplerosis. Beyond delineating these pathways, we critically evaluate emerging therapeutic paradigms that target these metabolic nodes, advocating for precision interventions capable of disrupting this pro-tumorigenic alliance while restoring immune surveillance.

Indexed as

cancerhypoxiametabolic reprogrammingtumor associated macrophagestumor microenvironment

Identifiers

PMID42100413
PMCPMC13143607

What Socratic holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.