Evidence map›Paper›PMID 42112403›Full record

ReviewFrontiers in immunology2026

TLR7 and TLR9 have overlapping but distinct roles in systemic lupus erythematosus and Sjögren's disease.

Sheta Biswas, Jill M Kramer

Abstract readReview
In one paragraph

Review in Frontiers in immunology, 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.

0numbers the graph read from it
0cells of the map it votes in
0citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

0 citing papers in PubMed.

No citing paper in PubMed yet.

4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

2 authors.

Sheta BiswasDepartment of Oral Biology, School of Dental Medicine, The University at Buffalo, State University of New York, Buffalo, NY, United States.
Jill M KramerDepartment of Oral Biology, School of Dental Medicine, The University at Buffalo, State University of New York, Buffalo, NY, United States.

Funding

Analysis of MyD88-mediated immune activation in Sjogrens syndrome pathogenesisR01DE029472 · NIDCR · STATE UNIVERSITY OF NEW YORK AT BUFFALO · PI Jill Marie Kramer · 2020 to 2026
$2.7M
NIDCR NIH HHS R01 DE029472
6 · The paper itself

Abstract

Endosomal toll-like receptors (TLRs), such as TLR7 and TLR9, are key mediators of autoimmunity. Systemic lupus erythematosus (SLE or lupus) and SjD are distinct diseases that are both characterized by heightened activation of TLR7 and TLR9 signaling networks that serve as potent modulators of chronic inflammation. This review will provide an overview of the role of these receptors in lupus and SjD, with a focus on recent mechanistic insights in the field. We will compare and contrast the roles of TLR7 and TLR9 in lupus and SjD, with a focus on the importance of B cell activation in disease. Moreover, we will discuss differences observed in sex-biased organ-specific disease manifestations. Finally, we will review current and emerging therapies that target endosomal TLR pathways and discuss their utility for treatment of SLE and SjD.

Indexed as

Lupus Erythematosus, SystemicSjogren's SyndromeToll-Like Receptor 7Toll-Like Receptor 9AnimalsAutoimmunityB-LymphocytesHumansLymphocyte ActivationSignal TransductionTLR7 protein, humanTLR9 protein, humanToll-Like Receptor 7Toll-Like Receptor 9age-associated B cellautoantibodiesplasmacytoid dendritic cell (pDC)salivary glandXIST

Identifiers

PMID42112403
PMCPMC13149362

What Socratic holds

Textmetadata
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Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.