Evidence mapPaperPMID 42158522Full record

ReviewInternational journal of nanomedicine2026

Unraveling Diabetes Mellitus-Driven Tumorigenesis: From Pathophysiological Mechanisms, Therapeutic Drugs to Tailor-Made Nanosystems.

Zhijie Jiang, Yongting Zhou, Mingfei Ma, Mingzhu Huang, Xiaojuan Wang

Abstract readReview
In one paragraph

Review in International journal of nanomedicine, 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.

0numbers the graph read from it
0cells of the map it votes in
0citing papers in PubMed
field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

0 citing papers in PubMed.

No citing paper in PubMed yet.

4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

5 authors.

Zhijie JiangDepartment of Clinical Pharmacy, The First Affiliated Hospital, Zhejiang University School of Medicine, Hangzhou, 310000, People's Republic of China.
Yongting ZhouDepartment of Clinical Pharmacy, The First Affiliated Hospital, Zhejiang University School of Medicine, Hangzhou, 310000, People's Republic of China.
Mingfei MaDepartment of Clinical Pharmacy, The First Affiliated Hospital, Zhejiang University School of Medicine, Hangzhou, 310000, People's Republic of China.
Mingzhu HuangDepartment of Clinical Pharmacy, The First Affiliated Hospital, Zhejiang University School of Medicine, Hangzhou, 310000, People's Republic of China.
Xiaojuan WangDepartment of Clinical Pharmacy, The First Affiliated Hospital, Zhejiang University School of Medicine, Hangzhou, 310000, People's Republic of China.

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Diabetes mellitus (DM) and cancer are two major global public health challenges. Growing evidence shows that the core features of DM, including hyperactivated insulin/IGF-1 signaling axis, chronic hyperglycemia, and a persistent inflammatory microenvironment, are important factors driving tumorigenesis and progression. Currently available clinical antidiabetic drugs, such as chemically synthesized drugs, biological and natural products, have been proven to achieve a dual therapeutic effect of controlling blood glucose and regulating the tumor microenvironment (TME). However, these drugs face practical difficulties such as poor oral bioavailability, off-target side effects, and low drug accumulation at tumor sites in clinical application. To overcome these challenges, nanotechnology offers a highly promising solution. Thus, this review elucidates the mechanisms of DM-driven cancer development and the anti-tumor therapeutic mechanisms of current anti-diabetic drugs. Then, we highlight how tailor-made nanosystems overcome traditional delivery barriers, offering unique advantages in targeted delivery, integrated diagnostics, and the oral administration of fragile biologics. Finally, the clinical translatability, disadvantages, and future prospects of these nanosystems will be briefly discussed.

Indexed as

CarcinogenesisDiabetes MellitusHypoglycemic AgentsNanoparticle Drug Delivery SystemNeoplasmsAnimalsAntineoplastic AgentsDrug Delivery SystemsHumansAntineoplastic AgentsHypoglycemic AgentsNanoparticle Drug Delivery Systemcancer developmentdiabetes mellitushyperglycemianano-delivery systemstherapeutic agents

Identifiers

PMID42158522
PMCPMC13182747

What Socratic holds

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LicenceCC BY-NC
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Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.