Evidence map›Paper›PMID 42175658›Full record

Observational studyUnited European gastroenterology journal2026

Molecular Traces of Gastric Cancer in Saliva: From Tissue Signatures to Salivary SLC5A5 as a Potential Biomarker.

Catarina Lopes, Andreia Brandão, Dimitris Vavoulis, Sofia Paulino, João Costa, Inês Marques de Sá, Sara Archer, Ricardo Küttner-Magalhães, Ricardo Marcos-Pinto, Diogo Libânio and 2 more

Abstract readObservational StudyValidation Study
In one paragraph

Observational study in United European gastroenterology journal, 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.

0numbers the graph read from it
0cells of the map it votes in
0citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

0 citing papers in PubMed.

No citing paper in PubMed yet.

4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

12 authors.

Catarina LopesPrecancerous Lesions and Early Cancer Management Group, Research Center of IPO Porto (CI-IPOP)/CI-IPOP@RISE (Health Research Group), Portuguese Oncology Institute of Porto (IPO Porto)/Porto Comprehensive Cancer Center Raquel Seruca (Porto.CCC), Porto, Portugal.
Andreia BrandãoCancer Genetics Group, Research Center of IPO Porto (CI-IPOP)/CI-IPOP@RISE (Health Research Group), Portuguese Oncology Institute of Porto (IPO Porto)/Porto Comprehensive Cancer Center Raquel Seruca (Porto.CCC), Porto, Portugal.ORCID https://orcid.org/0000-0003-0938-1543
Dimitris VavoulisDepartment of Oncology, Oxford Molecular Diagnostic Center, University of Oxford, Oxford, UK.
Sofia PaulinoDepartment of Pathology, Portuguese Oncology Institute of Porto, Porto, Portugal.
João CostaDepartment of Pathology, Portuguese Oncology Institute of Porto, Porto, Portugal.
Inês Marques de SáDepartment of Gastroenterology, Portuguese Oncology Institute of Porto, Porto, Portugal.
Sara ArcherCentro Hospitalar Universitário de Santo António, Porto, Portugal.
Ricardo Küttner-MagalhãesPrecancerous Lesions and Early Cancer Management Group, Research Center of IPO Porto (CI-IPOP)/CI-IPOP@RISE (Health Research Group), Portuguese Oncology Institute of Porto (IPO Porto)/Porto Comprehensive Cancer Center Raquel Seruca (Porto.CCC), Porto, Portugal.
Ricardo Marcos-PintoPrecancerous Lesions and Early Cancer Management Group, Research Center of IPO Porto (CI-IPOP)/CI-IPOP@RISE (Health Research Group), Portuguese Oncology Institute of Porto (IPO Porto)/Porto Comprehensive Cancer Center Raquel Seruca (Porto.CCC), Porto, Portugal.
Diogo LibânioPrecancerous Lesions and Early Cancer Management Group, Research Center of IPO Porto (CI-IPOP)/CI-IPOP@RISE (Health Research Group), Portuguese Oncology Institute of Porto (IPO Porto)/Porto Comprehensive Cancer Center Raquel Seruca (Porto.CCC), Porto, Portugal.
Mário Dinis-RibeiroPrecancerous Lesions and Early Cancer Management Group, Research Center of IPO Porto (CI-IPOP)/CI-IPOP@RISE (Health Research Group), Portuguese Oncology Institute of Porto (IPO Porto)/Porto Comprehensive Cancer Center Raquel Seruca (Porto.CCC), Porto, Portugal.
Carina PereiraPrecancerous Lesions and Early Cancer Management Group, Research Center of IPO Porto (CI-IPOP)/CI-IPOP@RISE (Health Research Group), Portuguese Oncology Institute of Porto (IPO Porto)/Porto Comprehensive Cancer Center Raquel Seruca (Porto.CCC), Porto, Portugal.

Funding

Fundação para a Ciência e a Tecnologia 2021.03835.CEECINDFundação para a Ciência e a Tecnologia 2023.14811.TENURE.006Fundação para a Ciência e a Tecnologia UI/BD/151488/2021HORIZON EUROPE Framework Program 101095359UK Research and Innovation 101101252
6 · The paper itself

Abstract

backgroundEarly detection of gastric cancer (GC) and reliable risk stratification for metachronous gastric lesions (MGLs) remain societal and clinical challenges, particularly in intermediate risk populations. Non-invasive approaches such as saliva-based biomarkers could complement current strategies. The aim of this study was to identify and validate a tissue-based gene expression signature for early gastric lesions, explore its potential for MGL prediction, and assess its detectability in saliva.

methodsThree studies were conducted: (1) a retrospective case control study to identify (RNA sequencing with machine learning) and validate (reverse transcription [RT] quantitative polymerase chain reaction [qPCR]) a gene expression signature for early gastric cancer using formalin-fixed paraffin-embedded (FFPE) samples, (2) a retrospective longitudinal study evaluating the ability of the signature to stratify MGL risk, and (3) a prospective study testing the signature in saliva using droplet digital (dd)PCR in patients with gastric lesions and endoscopy-confirmed controls.

resultsA six-gene tissue-based expression signature (ADAMTSL1, CCNA2, HSP90AB1, HSPD1, PSAPL1, and SLC5A5) robustly discriminated early gastric lesions from non-tumor mucosa (area under the curve (AUC) = 0.96 and 95% confidence interval [CI]: 0.94-0.99). Models tailored for MGL prediction, incorporating clinical variables, achieved moderate performance (AUC = 0.74 and 95% CI: 0.59-0.88). In saliva, only the SLC5A5 gene showed consistent dysregulation. When combined with age and sex, the model reached an AUC of 0.78 (95% CI: 0.69-0.88) for the non-invasive detection of early GC, with a positive predictive value of 0.69 and negative predictive value of 0.81.

conclusionThis study presents a validated tissue-based gene signature for early GC detection and exploratory MGL risk stratification. Salivary SLC5A5 shows potential as a non-invasive biomarker, though its utility requires further validation in dedicated saliva-based studies.

Indexed as

Biomarkers, TumorNeoplasms, Second PrimarySalivaSodium-Iodide SymportersStomach NeoplasmsAdultAgedAged, 80 and overArea Under CurveCase-Control StudiesEarly Detection of CancerFemaleGastric MucosaGene Expression ProfilingHumansLongitudinal StudiesBiomarkers, TumorSLC5A5 protein, humanSodium-Iodide Symportersbiomarkersgastric lesionsliquid biopsypersonalized medicine

Identifiers

PMID42175658
PMCPMC13239249

What Socratic holds

Textmetadata
LicenceCC BY
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Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.