Evidence mapPaperPMID 42200183Full record

ArticleFrontiers in physiology2026

Walras modulates sex-dependent endoplasmic reticulum stress in cardiomyopathy.

Francisco J Martinez-Amaro, Carlos Garcia-Padilla, Fernando Bonet, Elena Alonso-Villa, Rocio Toro, Houria Daimi, Eva Vargas, Ivan Hernandez, Borja Vilaplana-Martí, Ignacio Perez de Castro and 10 more

Erratum issuedAbstract read
In one paragraph

Article in Frontiers in physiology, 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. An erratum has been issued. Not yet cited in PubMed.

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0cells of the map it votes in
0citing papers in PubMed
field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

0 citing papers in PubMed.

No citing paper in PubMed yet.

4 · The record

Corrections and comments

5 · Who and what money

Authors and funding

20 authors.

Francisco J Martinez-Amaro *Cardiovascular Research Group, Department of Experimental Biology, University of Jaen, Jaen, Spain.
Carlos Garcia-Padilla *Cardiovascular Research Group, Department of Experimental Biology, University of Jaen, Jaen, Spain.
Fernando BonetDepartment of Medicine, School of Medicine, University of Cádiz (UCA), Cádiz, Spain.
Elena Alonso-VillaDepartment of Medicine, School of Medicine, University of Cádiz (UCA), Cádiz, Spain.
Rocio ToroDepartment of Medicine, School of Medicine, University of Cádiz (UCA), Cádiz, Spain.
Houria DaimiCardiovascular Research Group, Department of Experimental Biology, University of Jaen, Jaen, Spain.
Eva VargasSystems Biology Unit, Department of Experimental Biology, University of Jaen, Jaen, Spain.
Ivan HernandezInstituto de Salud Carlos III, Madrid, Spain.
Borja Vilaplana-MartíInstituto de Salud Carlos III, Madrid, Spain.
Ignacio Perez de CastroInstituto de Salud Carlos III, Madrid, Spain.
Laetitia BouchardAix-Marseille Univ, MMG, Inserm U1251, Marseille, France.
Fabien HubertAix-Marseille Univ, MMG, Inserm U1251, Marseille, France.
Francesca RochaisAix-Marseille Univ, MMG, Inserm U1251, Marseille, France.
Antoine MuchirSorbonne Université, INSERM U974, Institute of Myology, Center of Research in Myology, Paris, France.
Michaela VeliovaCentro Nacional de Investigaciones Cardiovasculares Carlos III, Madrid, Spain.
José Antonio EnriquezCentro Nacional de Investigaciones Cardiovasculares Carlos III, Madrid, Spain.
Alejandro SalgueroIntercellular Signaling in Cardiovascular Development and Disease Laboratory, Centro Nacional de Investigaciones Cardiovasculares Carlos III, Madrid, Spain.
Jose Luis De La PompaIntercellular Signaling in Cardiovascular Development and Disease Laboratory, Centro Nacional de Investigaciones Cardiovasculares Carlos III, Madrid, Spain.
Estefania Lozano-VelascoCardiovascular Research Group, Department of Experimental Biology, University of Jaen, Jaen, Spain.
Diego FrancoCardiovascular Research Group, Department of Experimental Biology, University of Jaen, Jaen, Spain.

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Introduction: Cardiovascular diseases (CVDs) are the leading cause of death globally, taking an estimated 17.9 million lives each year. Most heart cardiomyopathies result in an increased need for protein production that translates into an increased endoplasmic reticulum stress and therefore in the activation of the unfolded protein response pathway (UPR). The sustained activation of this pathway produces cell death and worsens the course of the disease. The role of lncRNAs in UPR signalling and their impact in several cardiomyopathies is beginning to be addressed. Methods: To conduct our study we have performed real time PCR (qPCR), immunochemistry (IMQ), SeaHorse mithocondrial activity, Western blot (WB), Mass spectrometry (MS) and cell viability analysis. Results: Our results demonstrate a sex-dependent regulation of Discussion: Our data demonstrate that

Indexed as

dilated cardiomyopathyERShypertrophic cardiomyopathylncRNAsUPRWalras

Identifiers

PMID42200183
PMCPMC13199116

What Socratic holds

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Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.