Evidence map›Paper›PMID 42204608›Full record

ArticleBiology of sex differences2026

Distinctive behavioral and neurochemical profile of social stress in male and female adolescent mice.

Ezequiel Monferrer, Rebeca Vidal, Tomás Aledón-Catalá, Michele Malaguarnera, Esther O'Shea, José Miñarro, M Isabel Colado, Marta Rodríguez-Arias

Abstract read
In one paragraph

Article in Biology of sex differences, 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.

0numbers the graph read from it
0cells of the map it votes in
0citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

0 citing papers in PubMed.

No citing paper in PubMed yet.

4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

8 authors.

Ezequiel Monferrer *Department of Psychobiology, Faculty of Psychology, Universitat de ValènciaRIAPAD RD24/0003/0004 Instituto de Salud Carlos III, Avda. Blasco Ibáñez 21, 7, Valencia, 46010, Spain.
Rebeca Vidal *Departament of Pharmacology and Toxicology, Faculty of Medicine, Universidad Complutense de Madrid (UCM), Instituto de Investigación Sanitaria Hospital 12 de Octubre (Imas12), Instituto Universitario de Investigación Neuroquímica (IUIN-UCM), Red de Investigación en Atención Primaria de Adicciones (RIAPAd-ISCIII), Madrid, Spain.
Tomás Aledón-CataláDepartment of Psychobiology, Faculty of Psychology, Universitat de ValènciaRIAPAD RD24/0003/0004 Instituto de Salud Carlos III, Avda. Blasco Ibáñez 21, 7, Valencia, 46010, Spain.
Michele MalaguarneraDepartment of Psychobiology, Faculty of Psychology, Universitat de ValènciaRIAPAD RD24/0003/0004 Instituto de Salud Carlos III, Avda. Blasco Ibáñez 21, 7, Valencia, 46010, Spain.
Esther O'SheaDepartament of Pharmacology and Toxicology, Faculty of Medicine, Universidad Complutense de Madrid (UCM), Instituto de Investigación Sanitaria Hospital 12 de Octubre (Imas12), Instituto Universitario de Investigación Neuroquímica (IUIN-UCM), Red de Investigación en Atención Primaria de Adicciones (RIAPAd-ISCIII), Madrid, Spain.
José MiñarroDepartment of Psychobiology, Faculty of Psychology, Universitat de ValènciaRIAPAD RD24/0003/0004 Instituto de Salud Carlos III, Avda. Blasco Ibáñez 21, 7, Valencia, 46010, Spain.
M Isabel ColadoDepartament of Pharmacology and Toxicology, Faculty of Medicine, Universidad Complutense de Madrid (UCM), Instituto de Investigación Sanitaria Hospital 12 de Octubre (Imas12), Instituto Universitario de Investigación Neuroquímica (IUIN-UCM), Red de Investigación en Atención Primaria de Adicciones (RIAPAd-ISCIII), Madrid, Spain.
Marta Rodríguez-AriasDepartment of Psychobiology, Faculty of Psychology, Universitat de ValènciaRIAPAD RD24/0003/0004 Instituto de Salud Carlos III, Avda. Blasco Ibáñez 21, 7, Valencia, 46010, Spain. marta.rodriguez@uv.es.

Funding

Generalitat Valenciana PROMETEO (CIPROM/2021/080)Instituto de Salud Carlos III RD24/0003/0004Ministerio de Ciencia e Innovación PID-2020-112672RB-I00; PI22/00427 and PID2024-150450OB-I00
6 · The paper itself

Abstract

backgroundAdolescence is a critical developmental period characterized by increased vulnerability to social stress, closely associated with the emergence of long-term neurobiological alterations. The present study aimed to investigate the short- and long-term effects of social stress during adolescence in mice, using the classical social defeat (SD) model in males and the vicarious social defeat (VSD) model in females, and examining the role of the kynurenine (KYN) pathway in resilience and susceptibility phenotypes.

methodsA total of 264 OF1 mice were exposed to SD or VSD between PND 26 and 35. Animals were classified as resilient or susceptible based on their performance in the social interaction test (SIT). Behavioral assessments evaluating anxiety- and depression-like behaviors, as well as locomotor activity, were conducted in both the short term (adolescence) and long term (adulthood). In addition, corticosterone levels and tryptophan metabolites were measured in multiple brain regions.

resultsBoth SD and VSD increased corticosterone levels in both sexes. Depending on the SIT ratio, stressed mice were classified as resilient or susceptible, with females showing a lower proportion of susceptibility than males. In the short term, all defeated male mice exhibited increased locomotor activity, while anxiety-like behaviors were only observed in resilient male animals. Less time spent in back grooming was only observed in defeated adolescent females (both resilient and susceptible). Long-term effects were more limited, with persistent alterations mainly related to anxiety in resilient female mice. Regarding the KYN pathway, females displayed higher kynurenine levels and increased KYN/tryptophan ratios across several brain regions compared with males. Notably, only resilient females showed reduced KYN levels in the striatum. Furthermore, susceptible male mice exhibited an increased serotonin/tryptophan ratio in the cerebellum, limbic forebrain, and striatum compared to control or resilient males.

conclusionsOverall, these findings indicate that adolescent social stress induces sex-dependent behavioral and neurochemical responses, with susceptibility/resilience associated with specific sex-dependent alterations in the KYN pathway. These results highlight potential biomarkers underlying adaptive responses to stress and may inform the pathophysiology of stress-related mental disorders.

Indexed as

Behavior, AnimalSex CharacteristicsSocial DefeatStress, PsychologicalAnimalsAnxietyBrainCorticosteroneFemaleKynurenineMaleMiceMice, Inbred C57BLSocial BehaviorTryptophanCorticosteroneKynurenineTryptophanAnxietyCorticosteroneDepressionFemaleKynurenineMiceSocial defeatTryptophanVicarious social defeat

Identifiers

PMID42204608
PMCPMC13404580

What Socratic holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.