ArticleiScience2026
Gastric cancer-secreted galectin-1 promotes peritoneal mesothelial-mesenchymal transition to prime peritoneal metastasis soil.
Article in iScience, 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.
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The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
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Authors and funding
9 authors.
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Abstract
Peritoneal metastasis (PM) is the most common cause of death in gastric cancer (GC) patients. Targeting the molecular mechanisms of PM in GC could reduce its incidence and improve clinical outcomes. Using clinical specimen analysis, lentivirus transfection, western blotting, RT-qPCR, immunofluorescence, cell adhesion assays, and a GC peritoneal metastasis (GCPM) animal model, we explored galectin-1's role in promoting peritoneal MMT (mesothelial-mesenchymal transition) and enhancing PM through
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Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.