Evidence mapPaperPMID 42258447Full record

Trial reportDiabetes care2026

Effect of Finerenone on Albuminuria in Type 1 Diabetes by Baseline HbA1c Level and Diabetes Duration: An Exploratory Analysis of the FINE-ONE Trial.

Jelle M Beernink, Hiddo J L Heerspink, Andreas L Birkenfeld, David Z I Cherney, Helen M Colhoun, Per-Henrik Groop, Linong Ji, Chantal Mathieu, Sylvia E Rosas, Katherine R Tuttle and 14 more

Abstract readRandomized Controlled Trial
In one paragraph

Trial report in Diabetes care, 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.

0numbers the graph read from it
0cells of the map it votes in
0citing papers in PubMed
field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

0 citing papers in PubMed.

No citing paper in PubMed yet.

4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

24 authors.

Jelle M BeerninkDepartment of Clinical Pharmacy and Pharmacology, University of Groningen, University Medical Center Groningen, Groningen, the Netherlands.ORCID 0000-0002-4615-9012
Hiddo J L HeerspinkDepartment of Clinical Pharmacy and Pharmacology, University of Groningen, University Medical Center Groningen, Groningen, the Netherlands.ORCID 0000-0002-3126-3730
Andreas L BirkenfeldInstitute for Diabetes Research and Metabolic Diseases, Helmholtz Centre Munich, University of Tübingen, Tübingen, Germany.ORCID 0000-0003-1407-9023
David Z I CherneyDivision of Nephrology, University Health Network, Toronto General Hospital, University of Toronto, Toronto, Ontario, Canada.ORCID 0000-0003-4164-0429
Helen M ColhounInstitute of Genetics and Cancer, College of Medicine and Veterinary Medicine, University of Edinburgh, Edinburgh, U.K.ORCID 0000-0002-8345-3288
Per-Henrik GroopDepartment of Nephrology, University of Helsinki and Helsinki University Hospital, Helsinki, Finland.ORCID 0000-0003-4055-6954
Linong JiDepartment of Endocrinology and Metabolism, Peking University People's Hospital, Peking University Diabetes Center, Beijing, China.ORCID 0000-0002-3262-2168
Chantal MathieuClinical and Experimental Endocrinology, KU Leuven, Leuven, Belgium.ORCID 0000-0002-6099-2406
Sylvia E RosasKidney and Hypertension Unit, Joslin Diabetes Center and Harvard Medical School, Boston, MA.ORCID 0000-0002-9903-4002
Katherine R TuttleProvidence Inland Northwest Health, University of Washington School of Medicine, Spokane, WA.ORCID 0000-0002-2235-0103
Jay S SkylerDepartment of Medicine and Diabetes Research Institute, University of Miami Miller School of Medicine, Miami, FL.ORCID 0000-0003-1136-8110
Antonio J AmorDiabetes Unit, Endocrinology and Nutrition Department, Institut d'Investigacions Biomèdiques August Pi Sunyer (IDIBAPS), Hospital Clínic de Barcelona, University of Barcelona, Barcelona, Spain.ORCID 0000-0001-9237-0903
M Luiza CaramoriDepartment of Endocrinology and Metabolism, Institute of Clinical Sciences, Cleveland Clinic Foundation, Cleveland, OH.
Paolo FiorinaDivision of Endocrinology, ASST Fatebenefratelli-Sacco and Universita degli Studi di Milano, Milan, Italy.
Niels JongsDepartment of Clinical Pharmacy and Pharmacology, University of Groningen, University Medical Center Groningen, Groningen, the Netherlands.
Uberto PagottoDivision of Endocrinology and Diabetes Prevention and Care, IRCCS Policlinico Sant'Orsola and Azienda Ospedaliero-Universitaria di Bologna, Bologna, Italy.
Emanuela SetolaCardio-metabolic and Clinical Trials Unit, IRCCS San Raffaele Hospital Scientific Institute, Milan, Italy.
Meike BrinkerCardiology and Nephrology Clinical Development, Bayer AG, Wuppertal, Germany.
Robert LawatscheckCardiology and Nephrology Clinical Development, Bayer AG, Berlin, Germany.
Julie RusselBayer PLC, Reading, U.K.
Patrick SchloemerClinical Statistics & Analytics, Bayer AG, Berlin, Germany.
Janet B McGillDivision of Endocrinology, Metabolism & Lipid Research, Washington University School of Medicine in St. Louis, St. Louis, MO.
Peter RossingSteno Diabetes Center Copenhagen, Herlev, Denmark.ORCID 0000-0002-1531-4294
FINE-ONE investigators*

Funding

Bayer AG
6 · The paper itself

Abstract

objectiveTo evaluate whether the efficacy and safety of finerenone varied by baseline hemoglobin A1c (HbA1c) level, a proxy of glycemic control, and diabetes duration in people with type 1 diabetes and chronic kidney disease (CKD). RESEARCH DESIGN AND

methodsAdults with type 1 diabetes, urinary albumin-to-creatinine ratio (UACR) 200 to <5,000 mg/g, and estimated glomerular filtration rate (eGFR) 25 to <90 mL/min/1.73 m2 were randomly assigned (one to one) to finerenone or placebo. UACR change from baseline over 6 months by baseline HbA1c and diabetes duration was analyzed.

resultsBaseline HbA1c was available for 240 of 242 participants; mean (SD) HbA1c, diabetes duration, and eGFR were 7.6% (1.1%; 60 [12] mmol/mol), 32.0 (14.2) years, and 58.9 (19.2) mL/min/1.73 m2, respectively. At 6 months, HbA1c (95% CI) remained unchanged (finerenone +0.03% [-0.14%, 0.20%]; placebo 0.00% [-0.12%, 0.11%]; between-group difference +0.04% [-0.17%, 0.24%]; P = 0.74). Over 6 months, median UACR decreased from 574.6 to 373.5 mg/g with finerenone and from 506.4 to 475.6 mg/g with placebo, corresponding to a -25% placebo-corrected change (95% CI -35%, -13%; P = 0.0001). Treatment effects were consistent across HbA1c tertiles (<7.1%, ≥7.1% to ≤8.1%, and >8.1%), with placebo-corrected UACR changes (95% CIs) of -17% (-40%, 13%), -18% (-39%, 10%), and -37% (-55%, -13%), respectively (P interaction = 0.41). Effects were similarly consistent across diabetes duration tertiles (P interaction = 0.70). Overall safety and incidence of hyperkalemia were similar across HbA1c tertiles.

conclusionsIn adults with type 1 diabetes and CKD, finerenone reduced UACR and was well tolerated irrespective of HbA1c level or diabetes duration.

Indexed as

AlbuminuriaDiabetes Mellitus, Type 1Glycated HemoglobinNaphthyridinesAdultFemaleGlomerular Filtration RateHumansMaleMiddle AgedfinerenoneGlycated HemoglobinNaphthyridines

Identifiers

PMID42258447
PMCPMC13385502

What Socratic holds

Textmetadata
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Registered trials

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Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.