Evidence mapPaperPMID 42278005Full record

ArticleExperimental physiology2026

Gyumin Kang, Eui-Cheol Shin, Jae Kyeom Kim, Young Jun Kim

Abstract read
In one paragraph

Article in Experimental physiology, 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 1 paper.

0numbers the graph read from it
0cells of the map it votes in
1citing papers in PubMed
field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

1 citing paper in PubMed.

  1. Experimental physiology · 2026
    Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

4 authors.

Gyumin KangDepartment of Food Regulatory Science, Korea University, Sejong, Republic of Korea.ORCID https://orcid.org/0000-0003-3756-0880
Eui-Cheol ShinDepartment of GreenBio Science (BK21), Gyeongsang National University, Jinju, Republic of Korea.
Jae Kyeom KimDepartment of Food and Biotechnology, Korea University, Sejong, Republic of Korea.
Young Jun KimDepartment of Food Regulatory Science, Korea University, Sejong, Republic of Korea.

Funding

Ministry of Food and Drug Safety RS-2026-25522525National Research Foundation of Korea NRF-2021S1A5B5A17054553
6 · The paper itself

Abstract

Aerobic exercise enhances physiological performance by activating metabolic systems, including glucose oxidation and fat metabolism. Cellular glucose predominantly flows through glycolysis (95-98%), whereas a smaller fraction (2-5%) enters the hexosamine biosynthetic pathway (HBP), generating UDP-N-acetylglucosamine. Excessive carbohydrate intake and inactivity expedite glucose redirection toward the HBP via mass-action mechanisms, which impair glucose uptake and insulin signalling. Although exercise intensity is a key determinant of fuel selection, with lipid oxidation predominating at moderate intensities (25-40%

Indexed as

Biosynthetic PathwaysHexosaminesOxygen ConsumptionPhysical Conditioning, AnimalAnimalsGlucoseInsulinMaleMetabolomicsMiceMice, Inbred C57BLGlucoseHexosaminesInsulinall‐out aerobic exerciseglucose metabolismhexosamine biosynthetic pathwayinsulin sensitivity

Identifiers

PMID42278005
PMCPMC13327345

What Socratic holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.