Evidence mapPaperPMID 42278191Full record

ArticleInternational journal of molecular sciences2026

Apigenin as a Multitarget Anticancer Agent: Coordinated Inhibition of EGFR/MAPK and PI3K/Akt Signaling in Hematologic Malignancies.

Hatice Terzi, Şeyma Taştemur, Ayşegül Öztürk, Neslihan Başgöz Karaguş, Mustafa Eymen Kontaş, Onur Mahmutoğlu, Ali Güngör, Mehmet Şencan

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Article in International journal of molecular sciences, 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.

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0cells of the map it votes in
0citing papers in PubMed
field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

0 citing papers in PubMed.

No citing paper in PubMed yet.

4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

8 authors.

Hatice TerziDepartment of Internal Medicine, Department of Hematology, Faculty of Medicine, Sivas Cumhuriyet University, 58140 Sivas, Türkiye.ORCID 0000-0003-3471-1305
Şeyma TaştemurDepartment of Internal Medicine, Faculty of Medicine, Sivas Cumhuriyet University, 58140 Sivas, Türkiye.
Ayşegül ÖztürkDepartment of Therapy and Rehabilitation, Vocational School of Health Services, Sivas Cumhuriyet University, 58140 Sivas, Türkiye.
Neslihan Başgöz KaraguşDepartment of Medical Genetics, Faculty of Medicine, Erciyes University, 38280 Kayseri, Türkiye.
Mustafa Eymen KontaşDepartment of Internal Medicine, Department of Hematology, Faculty of Medicine, Sivas Cumhuriyet University, 58140 Sivas, Türkiye.ORCID 0009-0007-4487-4219
Onur MahmutoğluDepartment of Family Medicine, Sivas Provincial Health Directorate, 58080 Sivas, Türkiye.
Ali GüngörLaboratory and Veterinary Health Program, Health Services Vocational School, Osmaniye Korkut Ata University, 80000 Osmaniye, Türkiye.ORCID 0009-0008-7985-0986
Mehmet ŞencanDepartment of Internal Medicine, Department of Hematology, Faculty of Medicine, Sivas Cumhuriyet University, 58140 Sivas, Türkiye.

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Hematologic malignancies are driven by dysregulated growth and survival signaling pathways that promote proliferation, treatment resistance, and disease progression. Naturally derived compounds targeting multiple oncogenic pathways with low toxicity have gained interest. Apigenin, a dietary flavonoid, shows anticancer activity in solid tumors, but its molecular effects in hematologic malignancies remain unclear. The antineoplastic effects of apigenin were evaluated in K562 (chronic myeloid leukemia) and DOHH2 (B-cell lymphoma) cell lines. Cell viability was assessed using the CCK-8 assay. L929 (mouse fibroblast) cells were included to evaluate selectivity. EGFR, MAPK, PI3K, NF-κB, caspase-3, and caspase-7 levels were measured by ELISA. Apoptosis and cell cycle distribution were analyzed by flow cytometry. Apigenin reduced cell viability in a dose-dependent manner, with IC

Indexed as

Antineoplastic AgentsApigeninHematologic NeoplasmsProto-Oncogene Proteins c-aktSignal TransductionAnimalsApoptosisCell Line, TumorCell ProliferationCell SurvivalErbB ReceptorsHumansK562 CellsMiceMitogen-Activated Protein KinasesNF-kappa BAntineoplastic AgentsApigeninErbB ReceptorsMitogen-Activated Protein KinasesNF-kappa BPhosphatidylinositol 3-KinasesProto-Oncogene Proteins c-aktapigeninapoptosisDOHH2growth signaling pathwayshematological malignanciesK562

Identifiers

PMID42278191
PMCPMC13256365

What Socratic holds

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Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.