Evidence mapPaperPMID 42283046Full record

SynthesisThe Pan African medical journal2026

Association of apolipoprotein E polymorphisms (E2, E3, E4) with type 2 diabetes: a meta-analysis of case-control studies.

Chaimae Zouine, Mohamed Amine Ikhanjal, Khaoula Errafii, Hicham Charoute, Meriem Es-Saadi, Fouad Benhnini, Nouha Messoudi, Bouchra Ghazi, Salsabil Hamdi

Abstract readMeta-AnalysisReview
In one paragraph

Synthesis in The Pan African medical journal, 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.

0numbers the graph read from it
0cells of the map it votes in
0citing papers in PubMed
field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

0 citing papers in PubMed.

No citing paper in PubMed yet.

4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

9 authors.

Chaimae ZouineVirology Environmental Health Laboratory, Institut Pasteur du Maroc, Casablanca, Morocco.
Mohamed Amine IkhanjalVirology Environmental Health Laboratory, Institut Pasteur du Maroc, Casablanca, Morocco.
Khaoula ErrafiiAfrican Genomic Center (AGC), University Mohamed VI Polytechnic, Bengurir, Morocco.
Hicham CharouteResearch Unit of Epidemiology, Biostatistics and Bioinformatics, Institut Pasteur du Maroc, Casablanca, Morocco.
Meriem Es-SaadiVirology Environmental Health Laboratory, Institut Pasteur du Maroc, Casablanca, Morocco.
Fouad BenhniniLaboratory of Cellular Signaling, Faculty of Sciences Meknes, Moulay Ismail University, Meknes, Morocco.
Nouha MessoudiVirology Environmental Health Laboratory, Institut Pasteur du Maroc, Casablanca, Morocco.
Bouchra GhaziImmunopathology-Immunotherapy-Immuno monitoring Laboratory, Faculty of Medicine, Mohammed VI University of Health and Sciences, Casablanca, Morocco.
Salsabil HamdiVirology Environmental Health Laboratory, Institut Pasteur du Maroc, Casablanca, Morocco.

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Type 2 diabetes (T2D) accounts for over 90% of all diabetes cases and results from the interaction of genetic and environmental factors. However, the association between apolipoprotein E (APOE) gene polymorphisms and T2D risk shows considerable variation across different populations. This meta-analysis aims to clarify the association between APOE genotypes and alleles (E2E2, E2E3, E2E4, E3E4, E4E4) and the risk of T2D. Additionally, it examines the association of demographic, clinical, and biochemical parameters with T2D risk in cases and controls. Relevant articles providing genotypic and allelic frequencies of APOE polymorphisms were sourced from Google Scholar, Web of Science, Science Direct, and PubMed databases. These articles focus on peer-reviewed human case-control studies published in English until February 27, 2024. Data on APOE polymorphisms, biochemical, and clinical parameters were extracted. Statistical tests were performed using Review Manager 4.3.1 with results expressed using ORs and 95% CIs. Publication bias and heterogeneity were assessed using the Q test and Egger regression analysis. Thirty-two studies involving 19644 participants. The statistical analysis showed that BMI, SBP, DBP, TC, and LDL-C could potentially indicate a higher risk of T2D in cases compared to controls. Significant associations with T2D were found for the APOE E4E4 genotype (OR =1.94, 95% CI= [1.16, 3.23], P = 0.01, I

Indexed as

Apolipoproteins EDiabetes Mellitus, Type 2AllelesCase-Control StudiesGene FrequencyGenetic Predisposition to DiseaseGenotypeHumansPolymorphism, GeneticRisk FactorsApoE protein, humanApolipoproteins EAlipoprotein E geneallelegenetic association studypolymorphismstype 2 diabetes

Identifiers

PMID42283046
PMCPMC13252076

What Socratic holds

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Registered trials

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Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.