Evidence map›Paper›PMID 42284244›Full record

ReviewJournal of vascular research2026

Hemodynamic Stress as a Cause of the High Prevalence of Arterial Disease in Women.

Almut Schreiber, Ulrich Barz, Helmut Barz

Abstract readReview
In one paragraph

Review in Journal of vascular research, 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.

0numbers the graph read from it
0cells of the map it votes in
0citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

0 citing papers in PubMed.

No citing paper in PubMed yet.

4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

3 authors.

Almut SchreiberMedical Center for Occupational Medicine and Social Hygiene, Fit for work, Dresden, Germany, almut.schreiber@fit-for-work.eu.
Ulrich BarzDepartment of Internal Medicine, Helios Klinikum Pirna, Pirna, Germany.
Helmut BarzDepartment of Neuropathology, Dietrich Bonhoeffer Hospital, Neubrandenburg, Germany.

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

backgroundIntracranial aneurysms, spontaneous coronary artery dissection, and fibromuscular dysplasia are significantly more common in women than in men, but the cause of these diseases and the cause of the predominance in women are unknown. It is also unclear whether and how genetic factors (genetic disposition or genetic pleiotropy) can influence the development of these diseases. A literature search in PubMed was conducted to identify the causes of the three arterial diseases. Particular attention was paid to the hemodynamic load on the arteries. SUMMARY: The imbalance of body fluids in pregnancy, menstruation, and premenstrual syndrome contributes to structural changes in the arterial wall and can promote arterial diseases. KEY MESSAGES: Increase in blood volume, high blood flow velocity, and edema of the vessel wall are factors that contribute to arterial diseases and can explain the predominance of some arterial diseases in women. These hemodynamic factors are to be distinguished from genetic causes, arteriosclerosis, or inflammatory diseases.

Indexed as

Coronary artery dissectionFibromuscular dysplasiaIntracranial aneurysmsMenstruationPregnancy

Identifiers

PMID42284244
PMCPMC13472596

What Socratic holds

Textmetadata
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.