Evidence mapPaperPMID 42296725Full record

ReviewTransfusion and apheresis science : official journal of the World Apheresis Association : official journal of the European Society for Haemapheresis2026

The resurgence of whole blood transfusion: Evaluating microcirculatory evidence in hemorrhagic trauma care: A review.

Elliot Widd, Carlos Munoz, Keisuke Tsuruta, Pedro Cabrales

Abstract readReview
In one paragraph

Review in Transfusion and apheresis science : official journal of the World Apheresis Association : official journal of the European Society for Haemapheresis, 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 1 paper.

0numbers the graph read from it
0cells of the map it votes in
1citing papers in PubMed
field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

1 citing paper in PubMed.

  1. Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

4 authors.

Elliot WiddDepartment of Bioengineering, University of California San Diego, 9500 Gilman Dr., La Jolla, CA 92093-0412, United States. Electronic address: ewidd@ucsd.edu.
Carlos MunozDepartment of Bioengineering, University of California San Diego, 9500 Gilman Dr., La Jolla, CA 92093-0412, United States.
Keisuke TsurutaDepartment of Bioengineering, University of California San Diego, 9500 Gilman Dr., La Jolla, CA 92093-0412, United States; Department of Emergency and Critical Care Medicine, Nara Medical University, 840 Shijo-cho, Kashihara, Nara 634-8521, Japan; Department of Chemistry, Nara Medical University, 88 Shijo-cho, Kashihara, Nara 634-0813, Japan.
Pedro CabralesDepartment of Bioengineering, University of California San Diego, 9500 Gilman Dr., La Jolla, CA 92093-0412, United States.

Funding

Training in Bioengineering Research and Technology Development in Cardiovascular in Cardiopulmonary Health and DiseaseT32HL160507 · UNIVERSITY OF CALIFORNIA, SAN DIEGO · 2025 to 2025
$311k
NHLBI NIH HHS T32 HL160507
6 · The paper itself

Abstract

During resuscitation from hemorrhagic shock, fresh whole blood remains the most effective treatment option. However, fresh whole blood is a limited resource, and its availability continues to decline. Thus, blood is preserved as whole blood or as individual components [e.g., plasma, platelets, and red blood cells (RBCs)] for transfusion practices. The quality of red blood cells during storage can be diminished, leading to a set of changes and consequences called storage lesion, resulting in the release of acellular hemoglobin and its degradation products (heme and iron). This acellular Hb causes vasoconstriction and reduces blood flow by oxidative injury and nitric oxide scavenging. Beyond biochemical effects, storage lesion alters blood rheology and RBC biomechanics, further compromising microcirculatory perfusion and oxygen delivery after transfusion.. These morphological changes impair oxygen transport, decrease cell deformability, shorten post‑transfusion circulation time, and disrupt microcirculatory blood flow. In this review, we analyze how different blood storage strategies and component therapies alter cellular biomechanics, hemorheology, and blood endothelial interactions, and how these changes impact the safety and efficacy of transfusions for hemorrhage resuscitation through the lens of microcirculatory perfusion. We also highlight current and emerging markers of storage lesion and microcirculatory function and discuss experimental biochemical approaches to extend shelf life and develop viable transfusion alternatives. While storage techniques for components continue to advance, and blood substitutes face present limitations, we conclude that the low-titer group O whole blood holds immediate promise for significantly improving transfusion medicine and resuscitation outcomes.

Indexed as

Blood PreservationBlood TransfusionHemorrhageMicrocirculationShock, HemorrhagicWounds and InjuriesHumansHemorrhageMicrocirculationStorage LesionWhole Blood

Identifiers

PMID42296725
PMCPMC13335765

What Socratic holds

Textmetadata
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Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.