ArticleFrontiers in endocrinology2026
Electroacupuncture ameliorates embryo implantation dysfunction in mice via miR-30c-5p.
Article in Frontiers in endocrinology, 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.
What it found
Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.
The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
The trial behind it
Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.
Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.
Who cites it
0 citing papers in PubMed.
No citing paper in PubMed yet.
Corrections and comments
PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.
Authors and funding
2 authors.
Funding
No grant is acknowledged in the PubMed record.
Abstract
Objective: To investigate the intrinsic mechanism by which electroacupuncture (EA) treats embryo implantation dysfunction in mice from the perspective of exosomal miRNAs. Methods: Human trophoblast cells (HTR-8/Svneo) were co-cultured Results: Communication existed between HTR-8/Svneo cells and endometrial cells, and co-culture with Ishikawa cells significantly promoted the proliferation and invasion of HTR-8/Svneo cells. Exosomes derived from both Ishikawa and HEC-1-A cells promoted HTR-8/Svneo cell proliferation and invasion. High-throughput sequencing showed that miR-30c-5p was expressed at a low level in Ishikawa cell-derived exosomes but at a high level in HEC-1-A cell-derived exosomes. Conclusion: EA may ameliorate embryo implantation dysfunction in mice by downregulating miR-30c-5p in the endometrium.
Indexed as
Identifiers
What Socratic holds
Registered trials
Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.