Evidence mapPaperPMID 42319232Full record

Observational studyEuropean stroke journal2026

Sex differences in aetiology of intracerebral haemorrhage and associated small vessel disease patterns.

Linda Fabisch, Hatice Ozkan, Philip S Nash, Wenpeng Zhang, Martina Locatelli, Yang Du, Larysa Panteleienko, Carola Tamm, Lena Obergottsberger, Melanie Haidegger and 8 more

Abstract readObservational Study
In one paragraph

Observational study in European stroke journal, 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.

0numbers the graph read from it
0cells of the map it votes in
0citing papers in PubMed
field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

0 citing papers in PubMed.

No citing paper in PubMed yet.

4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

18 authors.

Linda FabischDepartment of Neurology, Medical University of Graz, Auenbruggerplatz 22, 8036 Graz, Austria.ORCID 0009-0007-2720-6083
Hatice OzkanUCL Stroke Research Centre, Department of Translational Neuroscience and Stroke, University College London Queen Square Institute of Neurology, Queen Square, London WC1N 3BG, United Kingdom.
Philip S NashUCL Stroke Research Centre, Department of Translational Neuroscience and Stroke, University College London Queen Square Institute of Neurology, Queen Square, London WC1N 3BG, United Kingdom.ORCID 0000-0003-2428-1575
Wenpeng ZhangUCL Stroke Research Centre, Department of Translational Neuroscience and Stroke, University College London Queen Square Institute of Neurology, Queen Square, London WC1N 3BG, United Kingdom.
Martina LocatelliUCL Stroke Research Centre, Department of Translational Neuroscience and Stroke, University College London Queen Square Institute of Neurology, Queen Square, London WC1N 3BG, United Kingdom.
Yang DuUCL Stroke Research Centre, Department of Translational Neuroscience and Stroke, University College London Queen Square Institute of Neurology, Queen Square, London WC1N 3BG, United Kingdom.
Larysa PanteleienkoUCL Stroke Research Centre, Department of Translational Neuroscience and Stroke, University College London Queen Square Institute of Neurology, Queen Square, London WC1N 3BG, United Kingdom.
Carola TammDepartment of Neurology, Medical University of Graz, Auenbruggerplatz 22, 8036 Graz, Austria.
Lena ObergottsbergerDepartment of Neurology, Medical University of Graz, Auenbruggerplatz 22, 8036 Graz, Austria.
Melanie HaideggerDepartment of Neurology, Medical University of Graz, Auenbruggerplatz 22, 8036 Graz, Austria.
Markus KneihslDepartment of Neurology, Medical University of Graz, Auenbruggerplatz 22, 8036 Graz, Austria.
Gerit WünschInstitute for Medical Informatics, Statistics and Documentation, Medical University of Graz, Auenbruggerplatz 2, 8036 Graz, Austria.
Christian EnzingerDepartment of Neurology, Medical University of Graz, Auenbruggerplatz 22, 8036 Graz, Austria.
Robert J SimisterUCL Stroke Research Centre, Department of Translational Neuroscience and Stroke, University College London Queen Square Institute of Neurology, Queen Square, London WC1N 3BG, United Kingdom.
Hans R JägerNeuroradiological Academic Unit, Department of Translational Neuroscience and Stroke, UCL Queen Square Institute of Neurology, Queen Square, London WC1N 3BG, United Kingdom.
Thomas GattringerDepartment of Neurology, Medical University of Graz, Auenbruggerplatz 22, 8036 Graz, Austria.
David J WerringUCL Stroke Research Centre, Department of Translational Neuroscience and Stroke, University College London Queen Square Institute of Neurology, Queen Square, London WC1N 3BG, United Kingdom.
Simon Fandler-HöflerDepartment of Neurology, Medical University of Graz, Auenbruggerplatz 22, 8036 Graz, Austria.ORCID 0000-0001-9043-0378

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

introductionCauses of intracerebral haemorrhage (ICH), in particular cerebral small vessel disease (SVD), are a frequent subject of current research, yet the potential role of sex differences remains uncertain. Therefore, we aimed to investigate whether there are sex-related differences in aetiology, MRI features of SVD and risks of recurrent cerebrovascular events in patients with ICH. PATIENTS AND

methodsWe included patients from 2 large observational ICH study cohorts (London/UK, Graz/Austria) with available MRI. ICH aetiology was defined based on brain MRI, vascular imaging as well as clinical findings. Multivariable regression models were fitted to assess sex differences in aetiology, SVD MRI features and recurrent stroke events.

resultsWe identified 1043 patients (mean age 66 years, 58% male) with acute ICH. Males had a higher prevalence of cryptogenic ICH than females (aOR 1.53; 95% CI, 1.01-2.33). Males also had a higher rate of presence of any lacune (aOR 1.47; 95% CI, 1.11-1.94), severely enlarged perivascular spaces in the basal ganglia (aOR 1.45; 95% CI, 1.04-2.02) and presence of small asymptomatic diffusion-weighted imaging lesions (aOR 1.51; 95% CI, 1.06-2.15). There were no sex differences regarding recurrent ICH, incident ischaemic stroke or mortality. DISCUSSION AND

conclusionThe higher rate of presence of any lacune and enlarged perivascular spaces in men with ICH implies a higher severity of arteriolosclerosis. The mechanisms underlying the higher occurrence of cryptogenic ICH in men might include transient risk factors or incipient SVD.

Indexed as

Cerebral HemorrhageCerebral Small Vessel DiseasesSex CharacteristicsAgedAged, 80 and overFemaleHumansMagnetic Resonance ImagingMaleMiddle AgedRisk FactorsSex Factorsaetiologyintracerebral haemorrhagesexsmall vessel disease

Identifiers

PMID42319232
PMCPMC13280940

What Socratic holds

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Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.