ArticleFrontiers in medicine2026
PBMC-derived FGF, PDGF, VEGF and GM-CSF secretion in endometriosis: a case-control
Article in Frontiers in medicine, 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.
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The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
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6 authors.
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Abstract
Background: Endometriosis is a chronic inflammatory disease with immune dysregulation in which angiogenic, and hematopoietic mediators are thought to contribute to ectopic lesion establishment and persistence. Whether circulating immune cells are intrinsically primed to secrete higher levels of pro-angiogenic growth factors remains unclear. This study evaluated Methods: In a case-control design, women with laparoscopically and histopathologically confirmed endometriosis ( Results: Baseline secretion of FGF, PDGF, VEGF and GM-CSF by PBMCs did not differ significantly between women with endometriosis and controls after correction for multiple comparisons. PHA stimulation induced marked shifts in secretion profiles across participants- characterized by increases in FGF, PDGF and VEGF and a decrease in GM-CSF-but neither stimulated concentrations nor percent changes differed significantly between groups following false discovery rate adjustment. In univariate logistic regression analyses, none of the baseline growth-factor measures significantly predicted the presence of endometriosis. Conclusion: Under standardized
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