Evidence mapPaperPMID 42335141Full record

ArticlePloS one2026

Synergistic anticancer activity of antimicrobial peptide nisin and doxorubicin against breast cancer cells via modulation of membrane permeability.

Chanita Phetdee, Suwatjanee Naephrai, Malinee Pradain, Yanisa Panporm, Ubaid Ahmad, Panchika Prangkio

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Article in PloS one, 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.

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field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

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Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

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4 · The record

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PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

6 authors.

Chanita PhetdeeDepartment of Chemistry, Faculty of Science, Chiang Mai University, Chiang Mai, Thailand.
Suwatjanee NaephraiDepartment of Chemistry, Faculty of Science, Chiang Mai University, Chiang Mai, Thailand.
Malinee PradainDepartment of Chemistry, Faculty of Science, Chiang Mai University, Chiang Mai, Thailand.
Yanisa PanpormDepartment of Chemistry, Faculty of Science, Chiang Mai University, Chiang Mai, Thailand.ORCID https://orcid.org/0009-0006-1387-9677
Ubaid AhmadDepartment of Chemistry, Faculty of Science, Chiang Mai University, Chiang Mai, Thailand.
Panchika PrangkioDepartment of Chemistry, Faculty of Science, Chiang Mai University, Chiang Mai, Thailand.ORCID https://orcid.org/0000-0002-6376-7116

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Breast cancer is the most frequently diagnosed cancer and remains the leading cause of death among females. Despite the rapid advancement in cancer therapy, the development of more effective therapeutic strategies and anticancer agents with a new mode of action remains a critical challenge. Combination of therapeutic agents is an attractive approach to enhance drug efficacy. Nisin, a cationic antimicrobial peptide, has been reported for its cytotoxicity against some cancer cell lines via several mechanisms, particularly membrane disruption. Nisin can bind preferentially to the negatively charged phospholipids, causing pore formation in cell membranes. In this study, we demonstrated that nisin exhibited membrane permeabilization preferentially with anionic lipids using liposomal leakage assay. Moreover, anticancer activity of nisin and doxorubicin (DOX) was investigated against two breast cancer cell lines, MCF-7 and MDA-MB-231, using MTT assay. Nisin demonstrated cytotoxic effect against breast cancer MCF-7 and MDA-MB-231, with IC50 values of 5-8 µM, while exhibiting lower cytotoxicity toward normal cells. Based on Bliss independence analysis, the synergistic effect between nisin and DOX was markedly observed in MCF-7 when treated with 10 μM nisin and 1 μM DOX for 24-48 h treatment. Furthermore, as demonstrated by fluorescent-based high-content analysis, nisin clearly caused cell membrane permeability and promoted DOX-induced DNA damage in both cell lines. Flow cytometry with Annexin-V staining revealed that co-treatment of nisin and DOX significantly enhanced apoptosis, as compared to individual treatments, particularly in MCF-7 cells, suggesting a key mechanism of action for anticancer activity. Cationic nisin can interact with biological membrane and modulate membrane permeability and fluidity, consequently facilitating DOX entry, inducing apoptosis and DNA damage. Overall, this study demonstrates that the combination of nisin and DOX could offer a new therapeutic approach for breast cancer treatment with a reduced chemotherapeutic drug dosage.

Indexed as

Antineoplastic AgentsBreast NeoplasmsCell Membrane PermeabilityDoxorubicinNisinApoptosisCell Line, TumorDrug SynergismFemaleHumansMCF-7 CellsMDA-MB-231 CellsAntineoplastic AgentsDoxorubicinNisin

Identifiers

PMID42335141
PMCPMC13289944

What Socratic holds

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Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.