ArticlePlant, cell & environment2026
CCRG1 Acts as a Downstream Effector of the Clock Proteins CCA1 and LHY to Coordinate Growth, Flowering, and Senescence.
Article in Plant, cell & environment, 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 1 paper.
What it found
Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.
The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
The trial behind it
Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.
Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.
Who cites it
1 citing paper in PubMed.
- CCRG1 Acts as a Downstream Effector of the Clock Proteins CCA1 and LHY to Coordinate Growth, Flowering, and Senescence.Plant, cell & environment · 2026Article
Corrections and comments
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Authors and funding
8 authors.
Funding
Abstract
The circadian clock is an endogenous oscillator that generates ~24-h rhythms, enabling plants to anticipate and adapt to daily environmental cycles. E3 ubiquitin ligases are central regulators of protein turnover in signaling pathways, but their roles in the plant circadian system have yet to be fully elucidated. In this study, we find that the transcript of E3 ligase CCRG1 shows diurnal rhythmicity and sustained oscillation under continuous light in Arabidopsis thaliana. Molecular analyses demonstrate that the core clock components LATE ELONGATED HYPOCOTYL (LHY) and CIRCADIAN CLOCK ASSOCIATED 1 (CCA1) directly regulate CCRG1 transcription through promoter binding. The ccrg1 single mutants exhibit an increase in vegetative growth compared with the wild-type plants. The introduction of CCRG1 mutation in the cca1 lhy double mutants significantly suppresses the developmental defects, restoring vegetative biomass and alleviating premature flowering and leaf senescence. Furthermore, CCA1
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Registered trials
Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.