Evidence map›Paper›PMID 42353259›Full record

ReviewInternational journal of molecular sciences2026

The Potential Role of Vitamin D in

Joanna Robaczyńska, Milena Kiljańczyk, Maciej Maj, Adam Kiljańczyk, Tomasz Byrski, Cezary Cybulski, Izabela Janiuk, Jacek Gronwald, Jan Lubiński

Abstract readReview
In one paragraph

Review in International journal of molecular sciences, 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.

0numbers the graph read from it
0cells of the map it votes in
0citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

0 citing papers in PubMed.

No citing paper in PubMed yet.

4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

9 authors.

Joanna RobaczyńskaDepartment of Genetics and Pathology, International Hereditary Cancer Center, Pomeranian Medical University, ul. Unii Lubelskiej 1, 71-252 Szczecin, Poland.ORCID 0009-0007-8728-6927
Milena KiljańczykDepartment of Genetics and Pathology, International Hereditary Cancer Center, Pomeranian Medical University, ul. Unii Lubelskiej 1, 71-252 Szczecin, Poland.ORCID 0000-0001-8050-1507
Maciej MajDepartment of Histology and Embryology, Medical University of Warsaw, 02-004 Warsaw, Poland.ORCID 0009-0009-2698-6692
Adam KiljańczykDepartment of Genetics and Pathology, International Hereditary Cancer Center, Pomeranian Medical University, ul. Unii Lubelskiej 1, 71-252 Szczecin, Poland.ORCID 0009-0000-9398-3592
Tomasz ByrskiDepartment of Oncology and Chemotherapy, Pomeranian Medical University, 71-252 Szczecin, Poland.
Cezary CybulskiDepartment of Genetics and Pathology, International Hereditary Cancer Center, Pomeranian Medical University, ul. Unii Lubelskiej 1, 71-252 Szczecin, Poland.ORCID 0000-0002-2819-3057
Izabela JaniukDepartment of Histology and Embryology, Medical University of Warsaw, 02-004 Warsaw, Poland.ORCID 0000-0001-8695-8401
Jacek GronwaldDepartment of Genetics and Pathology, International Hereditary Cancer Center, Pomeranian Medical University, ul. Unii Lubelskiej 1, 71-252 Szczecin, Poland.ORCID 0000-0002-3643-2871
Jan LubińskiDepartment of Genetics and Pathology, International Hereditary Cancer Center, Pomeranian Medical University, ul. Unii Lubelskiej 1, 71-252 Szczecin, Poland.ORCID 0000-0003-2931-3003

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Vitamin D is a fat-soluble secosteroid essential for skeletal development and calcium homeostasis, but it also exerts pleiotropic effects on numerous biological processes via its active metabolites. Vitamin D metabolites act as steroid hormones that regulate cell-cycle progression, proliferation, differentiation, apoptosis, immune responses, and multiple intracellular signaling pathways. Moreover, they modulate the expression of genes involved in carcinogenesis. As circulating vitamin D levels are influenced by diet, fortified foods, and supplementation, they represent a potentially modifiable factor. Whether vitamin D status affects cancer risk or disease progression in carriers of pathogenic

Indexed as

BRCA1 ProteinBreast NeoplasmsVitamin DFemaleGenetic Predisposition to DiseaseHeterozygoteHumansReceptors, CalcitriolBRCA1 ProteinBRCA1 protein, humanReceptors, CalcitriolVitamin Dbreast cancerBReast CAncer gene 1ovarian cancervitamin Dvitamin D receptor

Identifiers

PMID42353259
PMCPMC13300645

What Socratic holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.