Evidence map›Paper›PMID 42386309›Full record

ReviewEuropean respiratory review : an official journal of the European Respiratory Society2026

Air pollution-induced airway epithelial barrier dysfunction in COPD: a narrative review of mechanisms and therapeutic implications.

Zhong He, Puxu Yang, Lina Shao, Xinhui Fang, Zhenkun Zhao, Wei Sun, Yuxiu Song, Ying Xu, Xiaoyu Zhao, Ziwen Ma and 3 more

Abstract readReview
In one paragraph

Review in European respiratory review : an official journal of the European Respiratory Society, 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.

0numbers the graph read from it
0cells of the map it votes in
0citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

0 citing papers in PubMed.

No citing paper in PubMed yet.

4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

13 authors.

Zhong HeDepartment of Pulmonary and Critical Care Medicine, Shengjing Hospital of China Medical University, Shenyang, China.
Puxu YangUltrasound Department, The First Affiliated Hospital of China Medical University, Shenyang, China.
Lina ShaoLiaoning Provincial Key Laboratory of Oral Disease, Department of Endodontics, School and Hospital of Stomatology, China Medical University, Shenyang, China.
Xinhui FangDepartment of Cardiovascular Medicine, The First Affiliated Hospital of China Medical University, Shenyang, China.
Zhenkun ZhaoPathology Department, The Fourth Affiliated Hospital of China Medical University, Shenyang, China.
Wei SunDepartment of Ultrasound, Shengjing Hospital of China Medical University, Shenyang, China.
Yuxiu SongDepartment of Rehabilitation Medicine, Shengjing Hospital of China Medical University, Shenyang, China.
Ying XuDepartment of Pulmonary and Critical Care Medicine, Shengjing Hospital of China Medical University, Shenyang, China.
Xiaoyu ZhaoDepartment of Pulmonary and Critical Care Medicine, Shengjing Hospital of China Medical University, Shenyang, China.
Ziwen MaChina Medical University, Shenyang, China.
Yuanyi YueDepartment of Gastroenterology, Shengjing Hospital of China Medical University, Shenyang, China.
Xueqing WangDepartment of Gastroenterology, Shengjing Hospital of China Medical University, Shenyang, China.
Qiang ZhangDepartment of Pulmonary and Critical Care Medicine, Shengjing Hospital of China Medical University, Shenyang, China cmusjzq2808@126.com.

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Air pollution is particularly harmful to people with chronic obstructive pulmonary disease (COPD). Exposure to air pollution from sources such as biomass burning leads to a unique COPD phenotype. This phenotype mainly shows airway damage, with less emphysema than typical smoking-related phenotypes. Across various COPD phenotypes, airway epithelial barrier dysfunction is a basic pathological mechanism. This narrative review summarises the current available evidence on how different types of airborne pollutants, including particulate matter (PM), ozone (O

Indexed as

Air PollutantsAir PollutionInhalation ExposureLungParticulate MatterPulmonary Disease, Chronic ObstructiveRespiratory MucosaAnimalsAntioxidantsHumansOxidative StressPhenotypeRisk FactorsAir PollutantsAntioxidantsParticulate Matter

Identifiers

PMID42386309
PMCPMC13320622

What Socratic holds

Textmetadata
LicenceCC BY-NC
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.