Evidence mapPaperPMID 42394535Full record

ArticleESC heart failure2026

Prognostic impact of chronotropic incompetence in transthyretin cardiac amyloidosis: a multicentre study.

Damiano Magrì, Nikita Ermolaev, Vincenzo Castiglione, Robin Willixhofer, Antonello Maruotti, Sara Corradetti, Alfonso Russo, Yu Fu Ferrari Chen, Christophe D J Capelle, Christina Kronberger and 10 more

Abstract readMulticenter Study
In one paragraph

Article in ESC heart failure, 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.

0numbers the graph read from it
0cells of the map it votes in
0citing papers in PubMed
field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

0 citing papers in PubMed.

No citing paper in PubMed yet.

4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

20 authors.

Damiano MagrìDepartment of Clinical and Molecular Medicine, Sant'Andrea Hospital, 'Sapienza' University of Rome, Via Giorgio Nicola Papanicolau, 00189 Rome, Italy.ORCID 0000-0002-0614-2961
Nikita ErmolaevDivision of Cardiology, Department of Internal Medicine II, Medical University of Vienna, Vienna, Austria.
Vincenzo CastiglioneHealth Science Interdisciplinary Center, Scuola Superiore Sant'Anna, Pisa, Italy.
Robin WillixhoferDivision of Cardiology, Department of Internal Medicine II, Medical University of Vienna, Vienna, Austria.
Antonello MaruottiDepartment of Public Health and Epidemiology, Khalifa University, Abu Dhabi, United Arab Emirates.ORCID 0000-0001-8377-9950
Sara CorradettiDepartment of Clinical and Molecular Medicine, Sant'Andrea Hospital, 'Sapienza' University of Rome, Via Giorgio Nicola Papanicolau, 00189 Rome, Italy.ORCID 0000-0002-6588-4906
Alfonso RussoDepartment of Economia, Statistica e Finanza 'Giovanni Anania'-University of Calabria, Cosenza, Italy.
Yu Fu Ferrari ChenHealth Science Interdisciplinary Center, Scuola Superiore Sant'Anna, Pisa, Italy.
Christophe D J CapelleDivision of Cardiology, Department of Internal Medicine II, Medical University of Vienna, Vienna, Austria.
Christina KronbergerDivision of Cardiology, Department of Internal Medicine II, Medical University of Vienna, Vienna, Austria.ORCID 0000-0002-3866-3710
Giuseppe VergaroHealth Science Interdisciplinary Center, Scuola Superiore Sant'Anna, Pisa, Italy.ORCID 0000-0002-7066-6006
Claudio PassinoHealth Science Interdisciplinary Center, Scuola Superiore Sant'Anna, Pisa, Italy.
Elisabetta SalvioniCentro Cardiologico Monzino, IRCCS, Milan, Italy.
Irene MattavelliCentro Cardiologico Monzino, IRCCS, Milan, Italy.ORCID 0000-0002-7781-1691
Arianna PiottiCentro Cardiologico Monzino, IRCCS, Milan, Italy.
Michele EmdinHealth Science Interdisciplinary Center, Scuola Superiore Sant'Anna, Pisa, Italy.
Emanuele BarbatoDepartment of Clinical and Molecular Medicine, Sant'Andrea Hospital, 'Sapienza' University of Rome, Via Giorgio Nicola Papanicolau, 00189 Rome, Italy.ORCID 0000-0002-0050-5178
Piergiuseppe AgostoniCentro Cardiologico Monzino, IRCCS, Milan, Italy.ORCID 0000-0003-2680-3867
Emiliano FioriDepartment of Clinical and Molecular Medicine, Sant'Andrea Hospital, 'Sapienza' University of Rome, Via Giorgio Nicola Papanicolau, 00189 Rome, Italy.ORCID 0000-0001-8749-8558
Roza Badr EslamDivision of Cardiology, Department of Internal Medicine II, Medical University of Vienna, Vienna, Austria.

Funding

DigiCardiopaTh PhD programPfizer 87737205Sapienza University of Rome
6 · The paper itself

Abstract

introductionChronotropic incompetence (CI) is a frequent but underappreciated feature of cardiac amyloidosis and may contribute to exercise intolerance. However, its prognostic significance remains incompletely defined. We investigated the prevalence, functional correlates, and prognostic value of chronotropic incompetence in patients with transthyretin amyloid cardiomyopathy.

methodsIn this multicentre retrospective study, 212 stable outpatients with transthyretin amyloid cardiomyopathy, in sinus rhythm and naïve to disease-specific therapy, underwent maximal cardiopulmonary exercise testing. Chronotropic response was assessed as peak heart rate expressed as a percentage of the age-predicted value (pHR%). Chronotropic incompetence prevalence was evaluated using clinically relevant thresholds. Prognostic performance for 1-year cardiovascular mortality was assessed using Cox regression, receiver operating characteristic analysis, and Kaplan-Meier estimates.

resultsChronotropic incompetence defined by a pHR% ≤75% was present in 35% of patients and was associated with markedly impaired functional capacity, including lower peak oxygen uptake and reduced ventilatory efficiency. During 1-year follow-up, 10 cardiovascular deaths occurred. Among exercise-derived variables, pHR% demonstrated strong prognostic value, with each 1% increase associated with a 5.5% relative reduction in cardiovascular mortality risk (hazard ratio 0.945; P = .011). A pHR% threshold of 75% provided optimal discrimination (area under the curve 0.71) and identified a subgroup with significantly lower survival (log-rank P < .05).

conclusionA blunted chronotropic response is common in transthyretin amyloid cardiomyopathy and conveys adverse short-term prognosis. A pHR% ≤75% represents a clinically meaningful and easily obtainable threshold for functional and prognostic stratification, offering a pragmatic alternative when comprehensive cardiopulmonary exercise testing assessment is not available.

Indexed as

Amyloid Neuropathies, FamilialCardiomyopathiesExercise ToleranceHeart RateAgedExercise TestFemaleFollow-Up StudiesHumansMalePrognosisRetrospective StudiesCardiac amyloidosisCardiopulmonary exercise testChronotropic incompetencePrognosis

Identifiers

PMID42394535
PMCPMC13344842

What Socratic holds

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Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.