Evidence map›Paper›PMID 42395903›Full record

ArticleFrontiers in microbiology2026

Feng-Yu Liu, Zhi-Zhen Liu, Lei Fang, Jing-Jing Huang, Xu-Hang Zhang, Xue-Ying Zhang, Chao-Nan Ma, Shan-Rui Shi, Hong-Hai Ji, Jun-Wei Xin and 1 more

Abstract read
In one paragraph

Article in Frontiers in microbiology, 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.

0numbers the graph read from it
0cells of the map it votes in
0citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

0 citing papers in PubMed.

No citing paper in PubMed yet.

4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

11 authors.

Feng-Yu Liu *Institute of Lipid Metabolism and Atherosclerosis, School of Pharmacy, Shandong Second Medical University, Weifang, China.
Zhi-Zhen Liu *School of Stomatology, Shandong Second Medical University, Weifang, China.
Lei Fang *School of Public Health, Shandong Second Medical University, Weifang, China.
Jing-Jing HuangInstitute of Lipid Metabolism and Atherosclerosis, School of Pharmacy, Shandong Second Medical University, Weifang, China.
Xu-Hang ZhangInstitute of Lipid Metabolism and Atherosclerosis, School of Pharmacy, Shandong Second Medical University, Weifang, China.
Xue-Ying ZhangInstitute of Lipid Metabolism and Atherosclerosis, School of Pharmacy, Shandong Second Medical University, Weifang, China.
Chao-Nan MaInstitute of Lipid Metabolism and Atherosclerosis, School of Pharmacy, Shandong Second Medical University, Weifang, China.
Shan-Rui ShiInstitute of Lipid Metabolism and Atherosclerosis, School of Pharmacy, Shandong Second Medical University, Weifang, China.
Hong-Hai JiSchool of Stomatology, Shandong Second Medical University, Weifang, China.
Jun-Wei XinInstitute of Lipid Metabolism and Atherosclerosis, School of Pharmacy, Shandong Second Medical University, Weifang, China.
Shou-Dong GuoInstitute of Lipid Metabolism and Atherosclerosis, School of Pharmacy, Shandong Second Medical University, Weifang, China.

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Background: While fucoidans show anti-atherosclerotic potential, their impact on this gut-host axis is unclear. This study investigated the effects of Methods: Atherosclerosis was induced in New Zealand rabbits via high-fat diet feeding combined with balloon catheter injury. Animals in the experimental groups ( Results: Treatments significantly reduced atherosclerotic plaque formation. This effect was particularly pronounced when fucoidan was combined with simvastatin, reducing plaque formation from 41.77 ± 16.02% to 5.91 ± 8.03% in the abdominal aorta and from 10.72 ± 3.49% to 2.29 ± 2.30% in the thoracic aorta (Model vs. combination group). The treatment also ameliorated hyperlipidemia, as shown by decreased plasma TC (24.55 ± 0.73 to 17.45 ± 0.58 mmol/L) and TG (7.75 ± 0.46 to 0.83 ± 0.25 mmol/L) in the combination group compared to the Model group. Both intervention groups exhibited enhanced microbial diversity and increased species richness across all taxonomic levels compared to the model group. Moreover, fucoidan and combination treatments significantly upregulated 83 and 128 metabolites and downregulated 125 and 121 metabolites, respectively. KEGG enrichment analysis indicated that these metabolic changes were associated with multiple pathways. Moreover, the combination therapy may mitigate certain side effects associated with simvastatin, although this possibility requires further investigation. Conclusion: The combination of

Indexed as

animal modelcardiovascular diseasefucoidangut metabolitesgut microbiotastatin

Identifiers

PMID42395903
PMCPMC13323129

What Socratic holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.