Evidence mapPaperPMID 42416583Full record

ReviewReviews in cardiovascular medicine2026

Integrin Signaling Pathways in Aortic Aneurysm and Dissection.

Ting Chen, Ping Li, Hongzhang Tong, Mengda Chen, Lingling Lu

Abstract readReview
In one paragraph

Review in Reviews in cardiovascular medicine, 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.

0numbers the graph read from it
0cells of the map it votes in
0citing papers in PubMed
field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

0 citing papers in PubMed.

No citing paper in PubMed yet.

4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

5 authors.

Ting ChenDepartment of Radiology, The Affiliated People's Hospital of Ningbo University, 315040 Ningbo, Zhejiang, China.ORCID https://orcid.org/0009-0007-6441-9571
Ping LiDepartment of Pathology, The Affiliated People's Hospital of Ningbo University, 315040 Ningbo, Zhejiang, China.ORCID https://orcid.org/0009-0005-6218-5092
Hongzhang TongDepartment of Radiology, The Affiliated People's Hospital of Ningbo University, 315040 Ningbo, Zhejiang, China.ORCID https://orcid.org/0009-0001-4697-528X
Mengda ChenDepartment of Radiology, The Affiliated People's Hospital of Ningbo University, 315040 Ningbo, Zhejiang, China.ORCID https://orcid.org/0009-0002-2037-7692
Lingling LuDepartment of Radiology, The Affiliated People's Hospital of Ningbo University, 315040 Ningbo, Zhejiang, China.ORCID https://orcid.org/0009-0001-0173-8977

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Aortic aneurysm (AA) and aortic dissection (AD) are life-threatening aortic diseases that primarily arise from medial degeneration. Ruptures are invariably associated with fatal outcomes; however, there are currently no clinically proven medications to prevent the progression of aortic aneurysm and dissection (AAD), and surgery remains the most effective approach to eliminating the high risk of aortic rupture. The dysfunction and depletion of vascular smooth muscle cells, along with inflammation and extracellular matrix (ECM) remodeling, are key mechanisms underlying AAD development. Integrins are heterodimeric transmembrane receptors that link the ECM to the cytoskeleton and transmit bidirectional cellular signals. Moreover, integrins maintain cell morphology and modulate numerous physiological and pathophysiological processes. Meanwhile, increasing evidence indicates that integrins play a vital role in AAD. This review outlines the basic biology of integrins, discusses the specific functions and mechanisms of integrins in AAD pathogenesis, and aims to identify reliable non-invasive biomarkers for acute AAD and potential targets to halt disease progression.

Indexed as

aortic aneurysmaortic dissectionintegrinsphysiologysignal transduction

Identifiers

PMID42416583
PMCPMC13339237

What Socratic holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.