Evidence mapPaperPMID 42423160Full record

ArticleCardiovascular therapeutics2026

Basement Membrane-Related Genes C1QTNF3, PTGER2, CAMK2N1, PRSS36, and B3GNT7: Novel Biomarkers for Coronary Artery Disease.

Zhao Dong, Yuyu Zhang, Zhuang Li, Haozhen Yu, Wei Ge

Abstract read
In one paragraph

Article in Cardiovascular therapeutics, 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.

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0citing papers in PubMed
field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

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Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

0 citing papers in PubMed.

No citing paper in PubMed yet.

4 · The record

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PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

5 authors.

Zhao DongDepartment of General Practice, Xijing Hospital, Fourth Military Medical University, Xi'an, Shaanxi, China, fmmu.edu.cn.ORCID https://orcid.org/0009-0000-1662-3067
Yuyu ZhangDepartment of General Practice, Xijing Hospital, Fourth Military Medical University, Xi'an, Shaanxi, China, fmmu.edu.cn.ORCID https://orcid.org/0009-0002-3074-4011
Zhuang LiDepartment of General Practice, Xijing Hospital, Fourth Military Medical University, Xi'an, Shaanxi, China, fmmu.edu.cn.ORCID https://orcid.org/0000-0003-2327-1316
Haozhen YuInstitute of Analytical Chemistry and Instrument for Life Science, the Key Laboratory of Biomedical Information Engineering of Ministry of Education, School of Life Science and Technology, Xi'an Jiaotong University, Xi'an, Shaanxi, China, xjtu.edu.cn.ORCID https://orcid.org/0009-0000-8549-2785
Wei GeDepartment of General Practice, Xijing Hospital, Fourth Military Medical University, Xi'an, Shaanxi, China, fmmu.edu.cn.ORCID https://orcid.org/0000-0002-9558-332X

Funding

National Natural Science Foundation of China 82270259
6 · The paper itself

Abstract

backgroundBasement membrane-related genes (BMRGs) play a pivotal role in the pathogenesis of several diseases. However, their association with coronary artery disease (CAD) remains unexplored. Therefore, this investigation is designed to elucidate the involvement of BMRGs in CAD mechanisms.

methodsThis study incorporated CAD-related datasets (GSE113079 and GSE125856) and BMRGs. Initially, differentially expressed genes (DEGs) were identified between CAD and control (CTL) groups in GSE113079 and GSE125856. Subsequently, weighted gene coexpression network analysis (WGCNA) was carried out to identify key module genes associated with basement membranes. We then intersected the DEGs with the key module genes to obtain differentially expressed basement membrane-related genes (DE-BMRGs). Two machine learning algorithms were applied to recognize feature genes, and the intersection of these features yielded the key genes. Subsequently, immune analysis, regulatory network construction, nomogram development, and expression verification were executed based on key genes.

resultsFive key genes were identified: C1QTNF3, PTGER2, CAMK2N1, PRSS36, and B3GNT7. Among these, C1QTNF3 (AUC = 0.954), PTGER2 (AUC = 0.878), CAMK2N1 (AUC = 0.872), and B3GNT7 (AUC = 0.855) exhibited strong diagnostic potential in GSE113079, whereas PRSS36 showed more modest performance (AUC = 0.667). Moreover, 11 differences were observed in immune cell populations between the two groups (p < 0.05), including CD8 T cells, and we constructed a TF-miRNA-gene network comprising 112 regulatory relationships, such as the regulation of C1QTNF3 by SMAD4 and hsa-miR-302a-3p. Additionally, the expression of these five key genes was consistently lower in the CAD group across GSE113079, GSE125856, and clinical patients.

conclusionThis study identified five key genes associated with the basement membrane (C1QTNF3, PTGER2, CAMK2N1, PRSS36, and B3GNT7) that offer valuable scientific insights into the mechanisms of CAD. These genes represent candidate noninvasive molecular markers requiring further validation for early diagnosis and serve as preliminary candidates for subsequent therapeutic target exploration.

Indexed as

Calcium-Calmodulin-Dependent Protein Kinase Type 2Coronary Artery DiseaseCase-Control StudiesDatabases, GeneticGene Expression ProfilingGene Regulatory NetworksGenetic MarkersGenetic Predisposition to DiseaseHumansReceptors, Prostaglandin E, EP4 SubtypeSignal TransductionCalcium-Calmodulin-Dependent Protein Kinase Type 2Genetic MarkersReceptors, Prostaglandin E, EP4 Subtypebasement membrane–related genescoronary artery diseasekey genes

Identifiers

PMID42423160
PMCPMC13347310

What Socratic holds

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Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.