Evidence map›Paper›PMID 42433452›Full record

ArticleRSC advances2026

Target deconvolution of a selective HepG2 cytotoxic hit identifies GSTP1 and TRAP1 as candidate molecular targets.

Belal O Al-Najjar, M Helal, Fadi G Saqallah, B Bandy

Abstract read
In one paragraph

Article in RSC advances, 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.

0numbers the graph read from it
0cells of the map it votes in
0citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

0 citing papers in PubMed.

No citing paper in PubMed yet.

4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

4 authors.

Belal O Al-NajjarDepartment of Pharmaceutical Sciences, Faculty of Pharmacy, Al-Ahliyya Amman University Amman 19328 Jordan b.najjar@ammanu.edu.jo.ORCID https://orcid.org/0000-0001-6811-1792
M HelalPhysiology, Pharmacology, and Toxicology Division, Biomedical Sciences Department, Faculty of Medicine and Health Sciences, An-Najah National University Nablus Palestine.
Fadi G SaqallahFaculty of Pharmacy, Al-Zaytoonah University of Jordan Amman 11733 Jordan.
B BandyCollege of Pharmacy and Nutrition, University of Saskatchewan Saskatoon SK S7N 5E4 Canada.

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Hepatocellular carcinoma (HCC) remains a lethal malignancy with limited therapeutic options in advanced disease, highlighting the need to identify selective cytotoxic agents and biologically relevant molecular targets for early anticancer drug discovery. In this study, a focused in-house compound set was screened against HepG2, MCF-7, and MDA-MB-231 cancer cell lines using the MTT assay to identify compounds with selective activity toward HCC-derived cells. The most active hit, HTS00019, was then subjected to an integrated computational target-deconvolution workflow comprising SEA target prediction, reverse docking against cancer-relevant proteins, binding-mode analysis, 100 ns molecular dynamics simulations, MM-GBSA binding free energy estimation, and

Identifiers

PMID42433452
PMCPMC13353288

What Socratic holds

Textmetadata
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.