ArticleThe Journal of neuroscience : the official journal of the Society for Neuroscience2026
Plasticity in Thoracic Paravertebral Sympathetic Postganglionic Neurons after High Spinal Cord Transection.
Article in The Journal of neuroscience : the official journal of the Society for Neuroscience, 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 1 paper.
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Who cites it
1 citing paper in PubMed.
- Satellite Glial Cells Drive Homeostatic Synaptic Structural Plasticity in Sympathetic Neurons.bioRxiv : the preprint server for biology · 2026Article
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6 authors.
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No grant is acknowledged in the PubMed record.
Abstract
Various presympathetic descending brain circuits recruit spinal cord preganglionic neurons to encode central sympathetic drive via their synaptic actions onto sympathetic postganglionic neurons (SPNs)-the final sympathetic output neurons. Thoracic paravertebral ganglia SPNs (tSPNs) provide distributed control over body tissue systems via functional subpopulations. High thoracic spinal cord injuries (SCIs) compromise supraspinal control of SPNs, causing dysautonomias including hypotension. In adult mice of either sex, we tested whether SCI-induced chronic loss of supraspinal control of tSPN activity leads to homeostatic increases in excitability. tSPN excitability spanned a >10-fold range in both sham and SCI populations, governed by a strong linear (ohmic) relationship between cell resistance and threshold depolarizing current (rheobase). The substantial variability obscured SCI-induced intrinsic plasticity. Dendritic length was reduced, as was measured cell capacitance in neuropeptide Y-expressing (NPY
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