Evidence mapPaperPMID 42447753Full record

ArticleEBioMedicine2026

Individualised treatment effects of corticosteroids in IgA nephropathy.

David L Hölscher, Nikolas E J Schmitz, Leon Niggemeier, Pourya Pilva, Martin Strauch, Vladimir Tesar, Jonathan Barratt, Ian S D Roberts, Rosanna Coppo, Laura Barisoni and 17 more

Abstract read
In one paragraph

Article in EBioMedicine, 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.

0numbers the graph read from it
0cells of the map it votes in
0citing papers in PubMed
field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

0 citing papers in PubMed.

No citing paper in PubMed yet.

4 · The record

Corrections and comments

5 · Who and what money

Authors and funding

27 authors.

David L HölscherInstitute of Pathology, RWTH Aachen University Hospital, Aachen, Germany; Department of Nephrology and Immunology, RWTH Aachen University Hospital, Aachen, Germany.
Nikolas E J SchmitzInstitute of Pathology, RWTH Aachen University Hospital, Aachen, Germany.
Leon NiggemeierInstitute of Pathology, RWTH Aachen University Hospital, Aachen, Germany.
Pourya PilvaInstitute of Pathology, RWTH Aachen University Hospital, Aachen, Germany.
Martin StrauchInstitute of Pathology, RWTH Aachen University Hospital, Aachen, Germany.
Vladimir TesarDepartment of Nephrology, 1st Faculty of Medicine and General University Hospital, Charles University, Prague, Czech Republic.
Jonathan BarrattJohn Walls Renal Unit, University Hospital of Leicester National Health Service Trust, Leicester, United Kingdom; Department of Cardiovascular Sciences, University of Leicester, Leicester, United Kingdom.
Ian S D RobertsDepartment of Cellular Pathology, Oxford University Hospitals National Health Service Foundation Trust, Oxford, United Kingdom.
Rosanna CoppoFondazione Ricerca Molinette, Torino, Italy; Regina Margherita Children's University Hospital, Torino, Italy.
Laura BarisoniDepartment of Pathology, Division of AI & Computational Pathology, Duke University, Durham, USA; Department of Medicine, Division of Nephrology, Duke University, Durham, USA.
Motoko YanagitaDepartment of Nephrology, Graduate School of Medicine, Kyoto University, Kyoto, Japan; Institute for the Advanced Study of Human Biology (WPI-ASHBi), Kyoto University, Kyoto, Japan.
Ulas AlabalikDepartment of Pathology, Medical Faculty, Dicle University, Diyarbakir, Turkey.
Andrew D RuleDivision of Nephrology and Hypertension, Mayo Clinic, Rochester, MN, USA; Division of Epidemiology, Mayo Clinic, Rochester, MN, USA.
Jaidip M JagtapDivision of Nephrology and Hypertension, Mayo Clinic, Rochester, MN, USA.
Eliabe S AbreuDivision of Nephrology and Hypertension, Mayo Clinic, Rochester, MN, USA.
Claudia SeikritDepartment of Nephrology and Immunology, RWTH Aachen University Hospital, Aachen, Germany.
Saskia von StillfriedInstitute of Pathology, RWTH Aachen University Hospital, Aachen, Germany.
Maarten W TaalCentre for Kidney Research and Innovation, University of Nottingham, Derby, United Kingdom; Renal Unit, University Hospitals of Derby and Burton NHS Foundation Trust, Derby, United Kingdom.
Philip A KalraDepartment of Renal Medicine, Salford Royal Hospital, Northern Care Alliance NHS Foundation Trust, Salford, United Kingdom.
Juergen FloegeDepartment of Nephrology and Immunology, RWTH Aachen University Hospital, Aachen, Germany; Department for Cardiology, RWTH Aachen University Hospital, Aachen, Germany.
Rafael KramannDepartment of Nephrology and Immunology, RWTH Aachen University Hospital, Aachen, Germany.
Peter BoorInstitute of Pathology, RWTH Aachen University Hospital, Aachen, Germany; Department of Nephrology and Immunology, RWTH Aachen University Hospital, Aachen, Germany. Electronic address: pboor@ukaachen.de.
Roman D BülowInstitute of Pathology, RWTH Aachen University Hospital, Aachen, Germany.
VALIGA investigators
CureGN investigators
NURTuRE academic steering group
AI4IgAN study group

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

backgroundIgA nephropathy (IgAN) has diverse clinical presentations and responses to treatment. For systemic corticosteroids in particular, randomised controlled trials have reported conflicting effects, highlighting the need for individualised treatment strategies.

methodsIn this retrospective cohort study, we derived and validated a causal machine learning (ML) framework to estimate individualised corticosteroid treatment effects in IgAN. Eight international cohorts, including the VALIGA, CureGN, and NURTuRE-CKD repositories, comprising 1022 patients, were analysed (derivation, n = 464; validation, n = 558). We integrated baseline clinical data, histopathological classification scores (MEST-C), and deep learning-based histomorphological biomarkers (pathomics) from digitised kidney biopsies. The framework estimated the effect of systemic corticosteroids on the composite endpoint of a ≥50% decline in estimated glomerular filtration rate or kidney failure within five years of biopsy.

findingsAcross the overall study population, systemic corticosteroid therapy was not associated with a significant improvement in the composite outcome (p = 0·27). However, the causal ML framework revealed substantial treatment heterogeneity, identifying patients with high predicted benefit who achieved longer progression-free survival with corticosteroids (0·43 years, 95% CI 0·18-0·73, p < 0·01), while no benefit was observed in those with low predicted benefit (-0·005 years, 95% CI -0·3 to 0·22, p > 0·05). An individualised framework-guided treatment assignment was estimated to reduce systemic corticosteroid use by 60·7%. Pathomics facilitated the identification of interstitial inflammation and tubulitis as key features of corticosteroid response.

interpretationThis study demonstrates that a causal ML framework integrating clinical, histopathological, and pathomics predictors can individualise treatment assignments for systemic corticosteroids in IgAN. This approach provides a blueprint for precision therapy in IgAN, supporting AI-enhanced clinical decision-making in the era of emerging targeted treatments.

fundingGerman Research Foundation; European Research Council; German Federal Ministry of Education and Research; German Innovation Fund of the Federal Joint Committee; Clinician Scientist Program of the Faculty of Medicine RWTH Aachen University.

Indexed as

Adrenal Cortex HormonesGlomerulonephritis, IGAPrecision MedicineAdultBiomarkersBiopsyFemaleGlomerular Filtration RateHumansMachine LearningMaleMiddle AgedRetrospective StudiesTreatment OutcomeAdrenal Cortex HormonesBiomarkersCausal machine learningComputational pathologyPathomicsPersonalised treatment

Identifiers

PMID42447753
PMCPMC13377490

What Socratic holds

Textmetadata
LicenceCC BY
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Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.