Evidence mapPaperPMID 42450646Full record

ArticleAnimals : an open access journal from MDPI2026

Eggshell Membrane Peptides Alleviate IL-1β-Induced Inflammatory Responses and Extracellular Matrix Degradation in Canine Chondrocytes by Inhibiting the NF-κB Signaling Pathway.

Xin Mao, Ling Xu, Yong Cao, Meifeng Wang, Wencan Wang

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Article in Animals : an open access journal from MDPI, 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.

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1 · What the graph read from it

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3 · Its place in the literature

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4 · The record

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5 · Who and what money

Authors and funding

5 authors.

Xin MaoPet Basic Research and Development Center, Chongqing Sweet Pet Products Co., Ltd., Chongqing 401120, China.
Ling XuPet Basic Research and Development Center, Chongqing Sweet Pet Products Co., Ltd., Chongqing 401120, China.
Yong CaoGuangdong Provincial Key Laboratory of Nutraceuticals and Functional Foods, College of Food Science, South China Agricultural University, Guangzhou 510642, China.
Meifeng WangChongqing Institute for Food and Drug Control, Chongqing 401121, China.
Wencan WangPet Basic Research and Development Center, Chongqing Sweet Pet Products Co., Ltd., Chongqing 401120, China.ORCID 0000-0002-8849-9090

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

backgroundEggshell membrane peptides (ESMPs) are natural bioactive compounds with reported chondroprotective properties. However, their regulatory effects on canine chondrocytes remain unclear. This study investigated ESMP in an interleukin-1β (IL-1β)-induced inflammatory model of canine chondrocytes.

methodsChondrocytes were assigned to control (Cont), IL-1β, and ESMP + IL-1β groups. Cell viability was assessed using the Cell Counting Kit-8 (CCK-8) assay. NF-κB p65 nuclear translocation was evaluated by immunofluorescence staining. Real-time quantitative PCR (RT-qPCR) and Western blotting (WB) were used to measure mRNA and protein expression levels, respectively.

resultsESMP inhibited IL-1β-induced NF-κB p65 nuclear translocation and reduced the IL-1β-induced increases in interleukin-6 (IL-6), cyclooxygenase-2 (COX-2), inducible nitric oxide synthase (iNOS), and matrix metalloproteinase-13 (MMP-13) at both mRNA and protein levels. ESMP also decreased IL-6, nitric oxide (NO), and prostaglandin E

conclusionsESMP attenuates IL-1β-induced inflammatory responses and extracellular matrix degradation in canine chondrocytes, potentially associated with suppression of NF-κB p65 nuclear translocation. This supports its potential application in promoting joint health in dogs.

Indexed as

canine chondrocyteseggshell membrane peptidesextracellular matrixIL-1βinflammatory responsesNF-κB signaling pathway

Identifiers

PMID42450646
PMCPMC13359944

What Socratic holds

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