ArticleFrontiers in immunology2026
Hematopoietic monoamine oxidase A deficiency exacerbates neuroinflammation and demyelination in female but not male mice with experimental autoimmune encephalomyelitis.
Article in Frontiers in immunology, 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.
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Abstract
Introduction: Monoamine oxidase A (Mao A) is a mitochondrial enzyme responsible for the degradation of monoaminergic neurotransmitters. While pharmacological inhibition of Mao A has been reported to improve outcomes in experimental autoimmune encephalomyelitis (EAE), the role of Mao A in immune cells on disease progression was unknown. Methods: To address this question, we generated mice with conditional Mao A deletion restricted to hematopoietic cells (Mao A Results and discussion: Mao A
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