Evidence map›Paper›PMID 42455481›Full record

ReviewMolecular diagnosis & therapy2026

Decoding and Overcoming Temozolomide Resistance Through CRISPR/Cas Technologies.

Bárbara S Marques, Maria Mendes, José Luís Alves, Alberto Pais, Carla Vitorino

Abstract readReview
PubMed Publisher
In one paragraph

Review in Molecular diagnosis & therapy, 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.

0numbers the graph read from it
0cells of the map it votes in
0citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

0 citing papers in PubMed.

No citing paper in PubMed yet.

4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

5 authors.

Bárbara S MarquesFaculty of Pharmacy, University of Coimbra, Coimbra, Portugal.
Maria MendesFaculty of Pharmacy, University of Coimbra, Coimbra, Portugal. mmendes@ff.uc.pt.
José Luís AlvesDepartment of Neurosurgery, ULS Coimbra, Coimbra, Portugal.
Alberto PaisCoimbra Chemistry Centre, Institute of Molecular Sciences - IMS, Department of Chemistry, University of Coimbra, Coimbra, Portugal.
Carla VitorinoFaculty of Pharmacy, University of Coimbra, Coimbra, Portugal. csvitorino@ff.uc.pt.ORCID http://orcid.org/0000-0003-3424-548X

Funding

Fundação para a Ciência e a Tecnologia 2022.06174.PTDCFundação para a Ciência e a Tecnologia UID/00313/2025Fundação para a Ciência e a Tecnologia UID/PRR/00313/2025
6 · The paper itself

Abstract

Intrinsic and acquired resistance to temozolomide (TMZ), the standard chemotherapeutic agent for glioblastoma (GBM), is highly common and results in poor clinical outcomes. This review highlights the emerging dual role of Clustered Regularly Interspaced Short Palindromic Repeats (CRISPR)-associated protein (Cas) technologies in addressing this challenge. First, it examines genome-wide CRISPR screens that revealed DNA damage repair networks, stress adaptations, stemness maintenance, and tumor heterogeneity as key drivers of resistance to TMZ. Second, it examines CRISPR/Cas-based strategies, including targeted gene disruption and epigenetic silencing of O

Identifiers

What Socratic holds

Textmetadata
LicenceCC BY-NC
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.